鉴定一种枯草芽孢杆菌spo0H等位基因，该基因对于抑制spo0A突变的孢子形成缺陷表型是必需的。
Identification of a Bacillus subtilis spo0H allele that is necessary for suppression of the sporulation-defective phenotype of a spo0A mutation.
作者信息
Bramucci M G, Green B D, Ambulos N, Youngman P
机构信息
Department of Microbiology and Immunology, Hahnemann University, Philadelphia, Pennsylvania 19102.
出版信息
J Bacteriol. 1995 Mar;177(6):1630-3. doi: 10.1128/jb.177.6.1630-1633.1995.
Abstract
A mutation in Bacillus subtilis spo0A codon 97 suppressed the sporulation defect caused by the spo0A9V mutation. The suppressor activity of the codon 97 mutation was evident only in the presence of a novel spo0H allele. Our results suggest that the spo0A gene product interacts with the sigma factor subunit of RNA polymerase.
摘要
枯草芽孢杆菌spo0A密码子97处的一个突变抑制了spo0A9V突变导致的芽孢形成缺陷。密码子97突变的抑制活性仅在存在一个新的spo0H等位基因时才明显。我们的结果表明，spo0A基因产物与RNA聚合酶的σ因子亚基相互作用。
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本文引用的文献
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Regulation of sigma B levels and activity in Bacillus subtilis.枯草芽孢杆菌中σB水平及活性的调控
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Bacillus sporulation gene spo0H codes for sigma 30 (sigma H).芽孢杆菌芽孢形成基因spo0H编码σ30（σH）。
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Characterization of the spo0A locus and its deduced product.spo0A基因座及其推导产物的特征分析。
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