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Leucocyte adhesion deficiency.

作者信息

el Habbal M H, Strobel S

机构信息

Cardiothoracic Unit, Institute of Child Health, London.

出版信息

Arch Dis Child. 1993 Oct;69(4):463-6. doi: 10.1136/adc.69.4.463.

DOI:10.1136/adc.69.4.463
PMID:7903143
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1029562/
Abstract
摘要

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本文引用的文献

1
Development of gene therapy for immunodeficiency: adenosine deaminase deficiency.免疫缺陷基因治疗的发展：腺苷脱氨酶缺乏症
Pediatr Res. 1993 Jan;33(1 Suppl):S49-53; discussion S53-5. doi: 10.1203/00006450-199305001-00278.
2
Recurrent infections and delayed separation of the umbilical cord in an infant with abnormal phagocytic cell locomotion and oxidative response during particle phagocytosis.一名婴儿在吞噬颗粒过程中存在吞噬细胞运动异常和氧化反应，出现反复感染及脐带延迟脱落。
J Pediatr. 1981 Dec;99(6):887-94. doi: 10.1016/s0022-3476(81)80011-x.
3
Subcellular localization of the large subunit of Mo1 (Mo1 alpha; formerly gp 110), a surface glycoprotein associated with neutrophil adhesion.与中性粒细胞黏附相关的表面糖蛋白Mo1（Mo1α；原称gp 110）大亚基的亚细胞定位。
J Clin Invest. 1984 Oct;74(4):1280-90. doi: 10.1172/JCI111538.
4
Deficiency of a surface membrane glycoprotein (Mo1) in man.人类表面膜糖蛋白（Mo1）缺乏症。
J Clin Invest. 1984 Jan;73(1):153-9. doi: 10.1172/JCI111186.
5
The severe and moderate phenotypes of heritable Mac-1, LFA-1 deficiency: their quantitative definition and relation to leukocyte dysfunction and clinical features.遗传性Mac-1、LFA-1缺陷的重度和中度表型：它们的定量定义及其与白细胞功能障碍和临床特征的关系。
J Infect Dis. 1985 Oct;152(4):668-89. doi: 10.1093/infdis/152.4.668.
6
Ligand binding by the p150,95 antigen of U937 monocytic cells: properties in common with complement receptor type 3 (CR3).U937单核细胞的p150,95抗原的配体结合：与3型补体受体（CR3）的共同特性。
Eur J Immunol. 1986 Sep;16(9):1117-23. doi: 10.1002/eji.1830160915.
7
LFA-1 immunodeficiency disease. Definition of the genetic defect and chromosomal mapping of alpha and beta subunits of the lymphocyte function-associated antigen 1 (LFA-1) by complementation in hybrid cells.淋巴细胞功能相关抗原1（LFA-1）免疫缺陷病。通过杂交细胞中的互补作用对淋巴细胞功能相关抗原1（LFA-1）的α和β亚基的遗传缺陷进行定义及染色体定位。
J Exp Med. 1986 Sep 1;164(3):855-67. doi: 10.1084/jem.164.3.855.
8
Periodontal manifestations of the heritable Mac-1, LFA-1, deficiency syndrome. Clinical, histopathologic and molecular characteristics.
J Periodontol. 1987 Jun;58(6):400-16. doi: 10.1902/jop.1987.58.6.400.
9
Increased surface expression of CD11b/CD18 (Mac-1) is not required for stimulated neutrophil adherence to cultured endothelium.中性粒细胞受刺激后黏附于培养的内皮细胞并不需要增加CD11b/CD18（巨噬细胞-1抗原）的表面表达。
J Clin Invest. 1988 Mar;81(3):676-82. doi: 10.1172/JCI113372.
10
Heterogeneity in the molecular defect leading to the leukocyte adhesion deficiency.导致白细胞黏附缺陷的分子缺陷的异质性。
Eur J Immunol. 1988 Oct;18(10):1575-9. doi: 10.1002/eji.1830181016.