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Involvement of tumor necrosis factor-alpha in glomerular injury.

作者信息

Baud L, Fouqueray B, Philipp C

机构信息

INSERM U. 64, Hôpital Tenon, Paris, France.

出版信息

Springer Semin Immunopathol. 1994;16(1):53-61. doi: 10.1007/BF00196713.

DOI:10.1007/BF00196713
PMID:7997946
Abstract
摘要

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2
Tumor necrosis factor alpha in glomerular injury.
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Mol Med. 1997 Feb;3(2):136-44.
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Mesangial cell-derived interleukin-10 modulates mesangial cell response to lipopolysaccharide.系膜细胞源性白细胞介素-10调节系膜细胞对脂多糖的反应。

本文引用的文献

1
TNF-alpha increases albumin permeability of isolated rat glomeruli through the generation of superoxide.肿瘤坏死因子-α通过超氧化物的产生增加离体大鼠肾小球的白蛋白通透性。
J Am Soc Nephrol. 1998 Mar;9(3):433-8. doi: 10.1681/ASN.V93433.
2
LPS-induced MCP-1, IL-1 beta, and TNF-alpha mRNA expression in isolated erythrocyte-perfused rat kidney.脂多糖诱导的分离的红细胞灌注大鼠肾脏中单核细胞趋化蛋白-1、白细胞介素-1β和肿瘤坏死因子-α信使核糖核酸的表达
Am J Physiol. 1993 May;264(5 Pt 2):F774-80. doi: 10.1152/ajprenal.1993.264.5.F774.
3
Thalidomide exerts its inhibitory action on tumor necrosis factor alpha by enhancing mRNA degradation.
Am J Pathol. 1995 Jul;147(1):176-82.
沙利度胺通过增强信使核糖核酸降解对肿瘤坏死因子α发挥抑制作用。
J Exp Med. 1993 Jun 1;177(6):1675-80. doi: 10.1084/jem.177.6.1675.
4
In situ production of TNF-alpha, IL-1 beta and IL-2R in ANCA-positive glomerulonephritis.抗中性粒细胞胞浆抗体（ANCA）阳性肾小球肾炎中肿瘤坏死因子-α（TNF-α）、白细胞介素-1β（IL-1β）和白细胞介素-2受体（IL-2R）的原位产生
Kidney Int. 1993 Mar;43(3):682-92. doi: 10.1038/ki.1993.98.
5
Elevated levels of tumor necrosis factor-alpha in the nephrotic syndrome in humans.人类肾病综合征中肿瘤坏死因子-α水平升高。
Am J Kidney Dis. 1993 Mar;21(3):251-9. doi: 10.1016/s0272-6386(12)80742-6.
6
Expression of IP-10, a lipopolysaccharide- and interferon-gamma-inducible protein, in murine mesangial cells in culture.脂多糖和γ干扰素诱导蛋白IP-10在培养的小鼠系膜细胞中的表达。
Am J Pathol. 1993 Feb;142(2):433-9.
7
Regulation of IFN-gamma and tumor necrosis factor-alpha expression in vivo. Effects of cycloheximide and cyclosporine in normal and lipopolysaccharide-treated mice.体内干扰素-γ和肿瘤坏死因子-α表达的调控。放线菌酮和环孢素对正常及脂多糖处理小鼠的影响。
J Immunol. 1993 Jan 15;150(2):342-52.
8
Oxygen radicals as second messengers for expression of the monocyte chemoattractant protein, JE/MCP-1, and the monocyte colony-stimulating factor, CSF-1, in response to tumor necrosis factor-alpha and immunoglobulin G. Evidence for involvement of reduced nicotinamide adenine dinucleotide phosphate (NADPH)-dependent oxidase.氧自由基作为单核细胞趋化蛋白JE/MCP-1和单核细胞集落刺激因子CSF-1表达的第二信使，以响应肿瘤坏死因子-α和免疫球蛋白G。还原型烟酰胺腺嘌呤二核苷酸磷酸（NADPH）依赖性氧化酶参与的证据。
J Clin Invest. 1993 Sep;92(3):1564-71. doi: 10.1172/JCI116737.
9
Effect of superoxide exposure on albumin permeability of isolated rat glomeruli.超氧化物暴露对分离的大鼠肾小球白蛋白通透性的影响。
J Lab Clin Med. 1993 Jun;121(6):797-804.
10
Role of tumor necrosis factor-alpha in the pathogenesis of glomerular diseases.
Kidney Int Suppl. 1993 Jan;39:S59-64.