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触觉失认症的本质:一项病例研究。

The nature of tactile agnosia: a case study.

作者信息

Reed C L, Caselli R J

机构信息

Department of Psychology, University of Pennsylvania, Philadelphia 19104-6196.

出版信息

Neuropsychologia. 1994 May;32(5):527-39. doi: 10.1016/0028-3932(94)90142-2.

DOI:10.1016/0028-3932(94)90142-2
PMID:8084412
Abstract

A chronic tactile agnosic with a small, MRI-documented left inferior parietal infarction underwent detailed somesthetic testing to assess (1) the acquisition of sensory data, (2) the manipulation of somatosensory percept and its association with previous knowledge, and (3) recognition occurring at a deeper taxonomic level. Results suggest that tactile agnosia can arise from faulty high-level perceptual processes, but that the ability to associate tactually defined objects and object parts with episodic memory can be preserved. Consistent with anatomic and physiologic studies in nonhuman primates, inferior parietal cortex (including Brodmann area 40, possibly area 39) appears to serve as a high-level somatosensory region.

摘要

一名患有慢性触觉失认症的患者,其左侧顶叶下部梗死灶经MRI证实较小,该患者接受了详细的躯体感觉测试,以评估:(1) 感觉数据的获取;(2) 躯体感觉知觉的操作及其与先前知识的关联;(3) 在更深分类水平上发生的识别。结果表明,触觉失认症可能源于错误的高级感知过程,但将触觉定义的物体和物体部分与情景记忆相关联的能力可能得以保留。与对非人类灵长类动物的解剖学和生理学研究一致,顶叶下部皮质(包括布罗德曼40区,可能还有39区)似乎是一个高级躯体感觉区域。

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