铅中毒大鼠肝脏中δ-氨基乙酰丙酸的变化 - Suppr | 超能文献

Changes in hepatic delta-aminolevulinic acid in lead-intoxicated rats.

作者信息

Suketa Y, Aoki M, Yamamoto T

出版信息

J Toxicol Environ Health. 1975 Sep;1(1):127-32. doi: 10.1080/15287397509529313.

DOI:10.1080/15287397509529313

Abstract

摘要

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Changes in hepatic delta-aminolevulinic acid in lead-intoxicated rats.铅中毒大鼠肝脏中δ-氨基乙酰丙酸的变化

J Toxicol Environ Health. 1975 Sep;1(1):127-32. doi: 10.1080/15287397509529313.

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Two methods for determining the activity of delta-aminolevulinate synthetase within intact liver cells in culture.两种用于测定培养的完整肝细胞内δ-氨基乙酰丙酸合成酶活性的方法。

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3

[Features of porphobilinogen synthesis from delta-aminolevulinic acid in the tissues of the internal organs of rats].

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Delta-aminolevulinic acid transport and synthesis, porphyrin synthesis and heme catabolism in chick embryo liver and heart cells.雏鸡胚胎肝脏和心脏细胞中的δ-氨基乙酰丙酸转运与合成、卟啉合成及血红素分解代谢

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Aminoketone synthesis in rat liver: age dependence of basic activity and inducibility by allyl isopropyl acetamide.大鼠肝脏中氨基酮的合成：基础活性的年龄依赖性以及烯丙基异丙基乙酰胺的诱导性

Acta Biol Med Ger. 1982;41(9):823-30.

6

[Biosynthesis of cytochrome c, III. Incorporation in vivo of radioactive delta-aminolevulinate, alpha-ketoglutarte, and succinicate into cytochrome c of the honey bee (author's transl)].细胞色素c的生物合成，III. 放射性δ-氨基乙酰丙酸、α-酮戊二酸和琥珀酸在蜜蜂体内掺入细胞色素c（作者译）

Hoppe Seylers Z Physiol Chem. 1973 Oct-Nov;354(10-11):1369-77.

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[Electron microscopic observations of liver and kidney of mous e treated with lead and ethanol, and variation of delta-ALA in urine and delta-ALA-D activities in blood (author's transl)].

Sangyo Igaku. 1977 Jul;19(4):196-7.

8

[Interrelation of delta-aminolevulinic acid and brain myelin proteins as affected by benzene and lead].[苯和铅对δ-氨基乙酰丙酸与脑髓磷脂蛋白相互关系的影响]

Biull Eksp Biol Med. 1985 Oct;100(10):447-9.

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Influence of allyl isopropyl acetamide on d-aminolevulinic acid and aminoacetone synthesis during postnatal development in rats.烯丙基异丙基乙酰胺对大鼠出生后发育过程中δ-氨基乙酰丙酸和氨基丙酮合成的影响。

Acta Biol Med Ger. 1982;41(9):831-5.

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[Porphobilinogen biosynthesis from delta-aminolevulinic acid by the internal organs of white rats].

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引用本文的文献

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Persistent protoporphyrinemia in hereditary porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency under low lead exposure. A new molecular basis for the pathogenesis of lead intoxication.低铅暴露下遗传性胆色素原合酶（δ-氨基-γ-酮戊酸脱水酶）缺乏所致的持续性原卟啉血症。铅中毒发病机制的一种新分子基础。

Klin Wochenschr. 1982 Jun 15;60(12):599-606. doi: 10.1007/BF01711435.

2

Acute lead poisoning in inherited porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency.遗传性胆色素原合酶（δ-氨基乙酰丙酸脱水酶）缺乏症中的急性铅中毒

Blut. 1982 Aug;45(2):131-9. doi: 10.1007/BF00319941.

3

Effects of delta-aminolaevulinic acid administration on social behaviour in the laboratory mouse.给予δ-氨基乙酰丙酸对实验小鼠社会行为的影响。

Psychopharmacology (Berl). 1979 Mar 22;61(2):131-5. doi: 10.1007/BF00426726.