Mounier-Vehier F, Leys D, Godefroy O, Rondepierre P, Marchau M, Pruvo J P
Department of Neurology, University Hospital, Lille, France.
Eur Neurol. 1994;34(1):11-5. doi: 10.1159/000117001.
Whether watershed infarcts and internal junctional infarcts have different mechanisms remains unknown. Of 493 consecutive patients with ischemic stroke or transient ischemic attack, 26 had 1 watershed infarct or more (8 anterior and 21 posterior) and 18 had 1 internal junctional infarct or more. Patients with watershed infarcts were more likely to have arterial hypertension [95% confidence intervals of odds ratio (CIOR): 1.04-6.15] and internal carotid artery stenosis > 50% (95% CIOR: 1.03-7.12) than patients without borderzone infarcts. Patients with internal junctional infarcts were more likely to have heart diseases than patients without borderzone infarcts (95% CIOR: 1.46-10.52). This preliminary study suggests that both subtypes of borderzone infarcts probably have different mechanisms.
分水岭梗死和内交界区梗死是否具有不同机制尚不清楚。在493例连续性缺血性卒中或短暂性脑缺血发作患者中，26例有1处或多处分水岭梗死（8例为前部，21例为后部），18例有1处或多处内交界区梗死。与无边缘带梗死的患者相比，分水岭梗死患者更易患动脉高血压[优势比的95%置信区间（CIOR）：1.04 - 6.15]和颈内动脉狭窄>50%（95% CIOR：1.03 - 7.12）。与无边缘带梗死的患者相比，内交界区梗死患者更易患心脏病（95% CIOR：1.46 - 10.52）。这项初步研究表明，边缘带梗死的两种亚型可能具有不同机制。
