Wenz R, Levine M R, Putterman A, Bersani T, Feldman K
Division of Ophthalmology, Mt. Sinai Medical Center, Cleveland, Ohio.
Ophthalmic Plast Reconstr Surg. 1994 Mar;10(1):34-41. doi: 10.1097/00002341-199403000-00007.
Three patients who had orbital decompression for compressive optic neuropathy secondary to Graves' ophthalmopathy are presented. Despite initial improvement in their visual acuity, all of the patients experienced subsequent relapse due to progressive enlargement of the extraocular muscles, resulting in recurrent compressive optic neuropathy. Management consisted of reoperation, systemic prednisone, and retrobulbar radiation. Enlargement of the extraocular muscles is felt to be caused by continuation of the autoimmune process and/or expansion of the extraocular muscles after slitting of the periorbita and bony removal of the orbital walls.
本文介绍了3例因格雷夫斯眼病继发压迫性视神经病变而接受眼眶减压术的患者。尽管他们的视力最初有所改善，但所有患者随后均因眼外肌逐渐增大而复发，导致复发性压迫性视神经病变。治疗方法包括再次手术、全身使用泼尼松和球后放疗。眼外肌增大被认为是自身免疫过程持续存在和/或眶周切开及眶壁骨质去除后眼外肌扩张所致。
