• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

支持血管性青光眼病因的论据。

Arguments for a vascular glaucoma etiology.

作者信息

Sonnsjö B, Krakau C E

机构信息

Department of Experimental Ophthalmology, University Eye Clinic, MAS, Malmö, Sweden.

出版信息

Acta Ophthalmol (Copenh). 1993 Aug;71(4):433-44. doi: 10.1111/j.1755-3768.1993.tb04615.x.

DOI:10.1111/j.1755-3768.1993.tb04615.x
PMID:8249571
Abstract

A material of Open Angle Glaucoma (G) collected by an eye practitioner (B S) from 1980 to 1991 is analysed with special respect to the occurrence of disc haemorrhages (Hs) and retinal vein occlusions (VOs). There is a close association between Hs and VOs and G, which in all categories increases with increasing follow-up time. Furthermore, the occurrence of mixed cases--H in one eye and VO in the same or the other--support the opinion that Hs, branch vein occlusions and central vein occlusions are manifestations of the same vascular disease, the difference between them being the size of the vessel affected. The well known morphological changes in the retinal veins of glaucomas and in central vein occlusions are endothelial proliferations causing progressive increase of flow resistance. Similar changes most probably also cause branch occlusions and disc haemorrhages. The vascular affection which obstructs the vessels with hindrance of the blood flow and impaired nutrition of neuronal tissue, might be the primary cause of glaucoma.

摘要

一位眼科医生(B S)在1980年至1991年期间收集的开角型青光眼(G)材料,特别针对视盘出血(Hs)和视网膜静脉阻塞(VOs)的发生情况进行了分析。Hs、VOs与G之间存在密切关联,在所有类别中,随着随访时间的增加而增加。此外,混合病例(一只眼为Hs,另一只眼或同一只眼为VO)的出现支持了这样一种观点,即Hs、分支静脉阻塞和中央静脉阻塞是同一血管疾病的表现,它们之间的差异在于受影响血管的大小。青光眼视网膜静脉和中央静脉阻塞中众所周知的形态学变化是内皮细胞增殖,导致血流阻力逐渐增加。类似的变化很可能也会导致分支阻塞和视盘出血。阻碍血管、妨碍血流并损害神经组织营养的血管病变可能是青光眼的主要原因。

相似文献

1
Arguments for a vascular glaucoma etiology.支持血管性青光眼病因的论据。
Acta Ophthalmol (Copenh). 1993 Aug;71(4):433-44. doi: 10.1111/j.1755-3768.1993.tb04615.x.
2
Disk hemorrhages and retinal vein occlusions in glaucoma.青光眼患者的视盘出血和视网膜静脉阻塞
Surv Ophthalmol. 1994 May;38 Suppl:S18-21; discussion S22. doi: 10.1016/0039-6257(94)90043-4.
3
Disc haemorrhages, precursors of open angle glaucoma.视盘出血,开角型青光眼的先兆。
Prog Retin Eye Res. 2002 Jan;21(1):35-56. doi: 10.1016/s1350-9462(01)00019-2.
4
Four cases of normal-tension glaucoma with disk hemorrhage combined with branch retinal vein occlusion in the contralateral eye.4例正常眼压性青光眼合并视盘出血,对侧眼合并视网膜分支静脉阻塞。
Am J Ophthalmol. 2004 Feb;137(2):357-9. doi: 10.1016/S0002-9394(03)00875-4.
5
Similarities between disc haemorrhages and thromboses of the retinal veins.视网膜静脉出血与血栓形成之间的相似性。
Int Ophthalmol. 1992 Sep;16(4-5):235-8. doi: 10.1007/BF00917967.
6
Optic disc haemorrhages and vascular abnormalities in a glaucoma population.青光眼患者人群中的视盘出血和血管异常
Aust N Z J Ophthalmol. 1997 May;25(2):137-44. doi: 10.1111/j.1442-9071.1997.tb01295.x.
7
Cup-to-disc ratio, intraocular pressure, and primary open-angle glaucoma in retinal venous occlusion.视网膜静脉阻塞中的杯盘比、眼压与原发性开角型青光眼
Ophthalmology. 2002 Feb;109(2):282-6. doi: 10.1016/s0161-6420(01)00922-8.
8
Primary angle closure and primary angle closure glaucoma in retinal vein occlusion.视网膜静脉阻塞中的原发性房角关闭和原发性房角关闭性青光眼。
Acta Ophthalmol. 2019 May;97(3):e364-e372. doi: 10.1111/aos.13879. Epub 2018 Sep 21.
9
Disc haemorrhages and the etiology of glaucoma.视盘出血与青光眼的病因
Acta Ophthalmol Suppl (1985). 1989;191:31-3. doi: 10.1111/j.1755-3768.1989.tb07084.x.
10
Retinal oximetry.视网膜血氧计。
Acta Ophthalmol. 2013 Mar;91 Thesis 2:1-47. doi: 10.1111/aos.12086.

引用本文的文献

1
Insights into modifiable risk factors of retinal vascular occlusion: A Mendelian randomization study.视网膜血管阻塞可改变危险因素的见解:一项孟德尔随机化研究。
Medicine (Baltimore). 2025 May 2;104(18):e41752. doi: 10.1097/MD.0000000000041752.
2
Intraocular pressure, primary open-angle glaucoma and the risk of retinal vein occlusion: A Mendelian randomization mediation analysis.眼内压、原发性开角型青光眼与视网膜静脉阻塞风险:孟德尔随机化中介分析。
Eye (Lond). 2024 Dec;38(17):3347-3351. doi: 10.1038/s41433-024-03303-x. Epub 2024 Aug 15.
3
Highlights on Genetic Polymorphism Associated with Thromboembolic Risk; Implications in Ophthalmic and Autoimmune Disorders-A Review.
与血栓栓塞风险相关的基因多态性研究亮点;对眼科和自身免疫性疾病的影响——综述
Diagnostics (Basel). 2023 Mar 27;13(7):1267. doi: 10.3390/diagnostics13071267.
4
Oxidative Stress and the Role of NADPH Oxidase in Glaucoma.氧化应激与NADPH氧化酶在青光眼中的作用
Antioxidants (Basel). 2021 Feb 4;10(2):238. doi: 10.3390/antiox10020238.
5
The impact of disc hemorrhage studies on our understanding of glaucoma: a systematic review 50 years after the rediscovery of disc hemorrhage.视盘出血研究对我们理解青光眼的影响:视盘出血重新发现50年后的系统评价
Jpn J Ophthalmol. 2019 Jan;63(1):7-25. doi: 10.1007/s10384-018-0641-2. Epub 2018 Nov 21.
6
Structural glaucomatous progression before and after occurrence of an optic disc haemorrhage.视盘出血发生前后的青光眼性结构进展
Br J Ophthalmol. 2015 Jan;99(1):21-5. doi: 10.1136/bjophthalmol-2014-305349. Epub 2014 Jul 23.
7
Morphology of optic disc through heidelberg retina tomograph in retinal vein occlusions alone or in combination with primary open angle glaucoma.单纯视网膜静脉阻塞或合并原发性开角型青光眼时,通过海德堡视网膜断层扫描仪观察视盘的形态学
Open Ophthalmol J. 2013 Jul 26;7:34-41. doi: 10.2174/1874364101307010034. eCollection 2013.
8
Comparison of myopic and nonmyopic disc hemorrhage in primary open-angle glaucoma.原发性开角型青光眼的近视性和非近视性盘出血比较。
Jpn J Ophthalmol. 2013 Mar;57(2):166-71. doi: 10.1007/s10384-012-0209-5. Epub 2012 Nov 27.
9
Recurrent disc hemorrhage does not increase the rate of visual field progression.反复性椎间盘出血并不会增加视野进展的速度。
Graefes Arch Clin Exp Ophthalmol. 2010 Jun;248(6):839-44. doi: 10.1007/s00417-010-1306-0. Epub 2010 Feb 25.
10
Disc hemorrhages in patients with both normal tension glaucoma and branch retinal vein occlusion in different eyes.双眼分别患有正常眼压性青光眼和视网膜分支静脉阻塞的患者的视盘出血。
Korean J Ophthalmol. 2007 Dec;21(4):222-7. doi: 10.3341/kjo.2007.21.4.222.