两性霉素B在体外和体内增强小鼠肿瘤坏死因子的产生。
Augmentation of murine tumor necrosis factor production by amphotericin B in vitro and in vivo.
作者信息
Tokuda Y, Tsuji M, Yamazaki M, Kimura S, Abe S, Yamaguchi H
机构信息
Department of Microbiology and Immunology, School of Medicine, Teikyo University, Tokyo, Japan.
出版信息
Antimicrob Agents Chemother. 1993 Oct;37(10):2228-30. doi: 10.1128/AAC.37.10.2228.
Abstract
Murine peritoneal macrophages were preincubated with amphotericin B (AMPH) and were then stimulated with bacterial lipopolysaccharide or streptococcal preparation (OK432). These macrophages produced a large amount of tumor necrosis factor. When administered to mice, the priming activity of amphotericin B for tumor necrosis factor production in vivo was also observed.
摘要
将两性霉素B（AMPH）与小鼠腹膜巨噬细胞预孵育，然后用细菌脂多糖或链球菌制剂（OK432）刺激这些巨噬细胞。这些巨噬细胞产生了大量肿瘤坏死因子。当将其给予小鼠时，还观察到两性霉素B在体内对肿瘤坏死因子产生的启动活性。
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