Semenets T N, Semina O V, Poverennyĭ A M, Deĭgin V I, Korotkov A M, Saenko A S
Radiats Biol Radioecol. 1993 Nov-Dec;33(6):812-5.
The data concerning the reasons of CFU-S radioresistance increase after low dose irradiation of mice were obtained; the reparation processes in CFU-S adapted by low dose irradiation were shown to be more active than in intact ones. Colony formation increase was demonstrated to be more pronounced in adapted animals when they had been injected by 50 mcg of synthetic polyribonucleotide PolyI-PolyC two days before irradiation in challenge dose. The thymogen immunomodulator administration to adapted and intact donor animals leads to the increase of colony yield in equal extent.
获得了关于小鼠低剂量照射后CFU-S抗辐射性增加原因的数据；结果表明，经低剂量照射适应后的CFU-S中的修复过程比未受照射的更为活跃。当在攻击剂量照射前两天给适应后的动物注射50微克合成多聚核糖核苷酸聚肌胞苷酸（PolyI-PolyC）时，适应后的动物中集落形成的增加更为明显。给适应后的和未受照射的供体动物施用胸腺原免疫调节剂会使集落产量同等程度增加。
