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Angiotensinogen: a candidate gene involved in preeclampsia?

作者信息

Arngrímsson R, Purandare S, Connor M, Walker J J, Björnsson S, Soubrier F, Kotelevtsev Y V, Geirsson R T, Björnsson H

出版信息

Nat Genet. 1993 Jun;4(2):114-5. doi: 10.1038/ng0693-114.

DOI:10.1038/ng0693-114
PMID:8348146
Abstract
摘要

相似文献

1
Angiotensinogen: a candidate gene involved in preeclampsia?血管紧张素原:子痫前期的一个候选基因?
Nat Genet. 1993 Jun;4(2):114-5. doi: 10.1038/ng0693-114.
2
The angiotensinogen gene M235T polymorphism and development of preeclampsia/eclampsia: a meta-analysis and meta-regression of observational studies.血管紧张素原基因M235T多态性与子痫前期/子痫的发生:观察性研究的荟萃分析和荟萃回归
J Hypertens. 2008 Sep;26(9):1726-34. doi: 10.1097/HJH.0b013e3283009ca5.
3
Multivariate analysis of genetic and acquired factors; T235 variant of the angiotensinogen gene is a potent independent risk factor for preeclampsia.遗传因素与后天因素的多变量分析;血管紧张素原基因的T235变体是先兆子痫的一个强大独立危险因素。
Semin Thromb Hemost. 2001;27(2):143-7. doi: 10.1055/s-2001-14073.
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Angiotensinogen gene variation in a population case-control study of preeclampsia/eclampsia in Australians and Chinese.澳大利亚人和中国人先兆子痫/子痫群体病例对照研究中的血管紧张素原基因变异
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Genetic analysis of the angiotensinogen gene in pre-eclampsia: study of german women and review of the literature.子痫前期血管紧张素原基因的遗传分析:德国女性研究及文献综述
Gynecol Obstet Invest. 2008;66(3):203-8. doi: 10.1159/000146084. Epub 2008 Jul 22.
6
Associations of polymorphisms of the angiotensinogen M235 polymorphism and angiotensin-converting-enzyme intron 16 insertion/deletion polymorphism with preeclampsia in Korean women.韩国女性中血管紧张素原M235多态性及血管紧张素转换酶内含子16插入/缺失多态性与子痫前期的相关性
Eur J Obstet Gynecol Reprod Biol. 2004 Sep 10;116(1):48-53. doi: 10.1016/j.ejogrb.2004.01.035.
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Genetic polymorphisms in vasoactive genes and preeclampsia: a meta-analysis.血管活性基因中的遗传多态性与子痫前期:一项荟萃分析。
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Molecular and genetic aspects of preeclampsia: state of the art.
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[Association of pregnancy-induced hypertension with a molecular variant of angiotensinogen gene].妊娠高血压与血管紧张素原基因分子变异的关联
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Evidence for an association of the R485K polymorphism in the coagulation factor V gene with severe preeclampsia from screening 35 polymorphisms in 27 candidate genes.通过对27个候选基因中的35个多态性进行筛查,发现凝血因子V基因R485K多态性与重度子痫前期之间存在关联的证据。
Thromb Haemost. 2001 Dec;86(6):1594-5.

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Genetic variation in placental insufficiency: What have we learned over time?胎盘功能不全中的基因变异:随着时间的推移我们学到了什么?
Front Cell Dev Biol. 2022 Oct 14;10:1038358. doi: 10.3389/fcell.2022.1038358. eCollection 2022.
2
AGT M235t polymorphism and heart failure in a cohort of Tunisian population: diagnostic and prognostic value.突尼斯人群队列中AGT M235t多态性与心力衰竭:诊断和预后价值
Int J Clin Exp Med. 2015 Sep 15;8(9):16346-51. eCollection 2015.
3
Association between Angiotensinogen M235T Polymorphism and Preeclampsia in Iranian Pregnant Women.
伊朗孕妇血管紧张素原M235T基因多态性与子痫前期的关联
J Family Reprod Health. 2014 Dec;8(4):169-73.
4
Variable effects of maternal and paternal-fetal contribution to the risk for preeclampsia combining GSTP1, eNOS, and LPL gene polymorphisms.结合谷胱甘肽S-转移酶P1(GSTP1)、内皮型一氧化氮合酶(eNOS)和脂蛋白脂肪酶(LPL)基因多态性,母体和父体-胎儿因素对先兆子痫风险的可变影响。
J Matern Fetal Neonatal Med. 2011 Apr;24(4):628-35. doi: 10.3109/14767058.2010.511351. Epub 2010 Sep 14.
5
Association of Angiotensin-converting enzyme and angiotensinogen gene polymorphisms with preeclampsia.血管紧张素转换酶及血管紧张素原基因多态性与子痫前期的关联
J Korean Med Sci. 2004 Apr;19(2):253-7. doi: 10.3346/jkms.2004.19.2.253.
6
Susceptibility loci for preeclampsia on chromosomes 2p25 and 9p13 in Finnish families.芬兰家族中位于2号染色体2p25和9号染色体9p13区域的子痫前期易感性基因座。
Am J Hum Genet. 2003 Jan;72(1):168-77. doi: 10.1086/345311. Epub 2002 Dec 9.
7
Hypertension in pregnancy: new recommendations for management.妊娠期高血压:管理新建议
Curr Hypertens Rep. 1999 Dec;1(6):529-39. doi: 10.1007/s11906-996-0026-7.
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Angiotensinogen variants and human hypertension.血管紧张素原变体与人类高血压。
Curr Hypertens Rep. 1999 Feb-Mar;1(1):31-41. doi: 10.1007/s11906-999-0071-0.
9
The preeclampsia enigma and the renin-angiotensin system.
J Mol Med (Berl). 2000;78(2):63-5. doi: 10.1007/s001090000087.
10
[Role of the angiotensinogen gene for essential hypertension].[血管紧张素原基因在原发性高血压中的作用]
Herz. 2000 Feb;25(1):15-25. doi: 10.1007/BF03044120.