肌萎缩侧索硬化症、超氧化物歧化酶与过氧亚硝酸盐 - Suppr

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ALS, SOD and peroxynitrite.

作者信息

Beckman J S, Carson M, Smith C D, Koppenol W H

出版信息

Nature. 1993 Aug 12;364(6438):584. doi: 10.1038/364584a0.

DOI:10.1038/364584a0

PMID:8350919

Abstract

摘要

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Unfolding and folding kinetics of amyotrophic lateral sclerosis-associated mutant Cu,Zn superoxide dismutases.

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ALS, SOD and peroxynitrite.

作者信息

Beckman J S, Carson M, Smith C D, Koppenol W H

出版信息

Nature. 1993 Aug 12;364(6438):584. doi: 10.1038/364584a0.

DOI:10.1038/364584a0

PMID:8350919

Abstract

摘要

相似文献

ALS, SOD and peroxynitrite.

Nature. 1993 Aug 12;364(6438):584. doi: 10.1038/364584a0.

[Amyotrophic lateral sclerosis and superoxide dismutase--a review].

Ugeskr Laeger. 1997 Mar 10;159(11):1593-6.

Manganese superoxide dismutase levels are elevated in a proportion of amyotrophic lateral sclerosis patient cell lines.

Biochem Biophys Res Commun. 2000 Jun 24;273(1):359-63. doi: 10.1006/bbrc.2000.2933.

Ann N Y Acad Sci. 2005 May;1042:303-13. doi: 10.1196/annals.1338.053.

[The role of SOD in pathogenesis amyotrophic lateral sclerosis].

Biull Eksp Biol Med. 1999 Apr;127(4):460-2.

Superoxide dismutase and neurofilament transgenic models of amyotrophic lateral sclerosis.

J Exp Zool. 1998;282(1-2):32-47.

Is ALS caused by an altered oxidative activity of mutant superoxide dismutase?

Nat Neurosci. 2002 Oct;5(10):919; author reply 919-20. doi: 10.1038/nn1002-919a.

The metabolic hypothesis in amyotrophic lateral sclerosis: insights from mutant Cu/Zn-superoxide dismutase mice.

Biomed Pharmacother. 2005 May;59(4):190-6. doi: 10.1016/j.biopha.2005.03.003. Epub 2005 Mar 17.

Brain superoxide dismutase, catalase, and glutathione peroxidase activities in amyotrophic lateral sclerosis.

Ann Neurol. 1996 Feb;39(2):158-65. doi: 10.1002/ana.410390204.

Unfolding and folding kinetics of amyotrophic lateral sclerosis-associated mutant Cu,Zn superoxide dismutases.

J Mol Biol. 2009 Jan 9;385(1):278-98. doi: 10.1016/j.jmb.2008.10.003. Epub 2008 Oct 11.

引用本文的文献

Alkaloids as neuroprotectors: targeting signaling pathways in neurodegenerative diseases.

Mol Cell Biochem. 2025 Apr 7. doi: 10.1007/s11010-025-05258-3.

Oxidative/Nitrosative Stress, Apoptosis, and Redox Signaling: Key Players in Neurodegenerative Diseases.

J Biochem Mol Toxicol. 2025 Jan;39(1):e70133. doi: 10.1002/jbt.70133.

Nuclear Localization of Human SOD1 in Motor Neurons in Mouse Model and Patient Amyotrophic Lateral Sclerosis: Possible Links to Cholinergic Phenotype, NADPH Oxidase, Oxidative Stress, and DNA Damage.

Int J Mol Sci. 2024 Aug 22;25(16):9106. doi: 10.3390/ijms25169106.

Crosstalk between Oxidative Stress and Inflammation Caused by Noise and Air Pollution-Implications for Neurodegenerative Diseases.

Antioxidants (Basel). 2024 Feb 22;13(3):266. doi: 10.3390/antiox13030266.

Cell Transplant. 2024 Jan-Dec;33:9636897241237049. doi: 10.1177/09636897241237049.

Mitochondria: A Promising Convergent Target for the Treatment of Amyotrophic Lateral Sclerosis.

Cells. 2024 Jan 29;13(3):248. doi: 10.3390/cells13030248.

Functions of Astrocytes under Normal Conditions and after a Brain Disease.

Int J Mol Sci. 2023 May 8;24(9):8434. doi: 10.3390/ijms24098434.

Oxidative Stress and Antioxidants in Neurodegenerative Disorders.

Antioxidants (Basel). 2023 Feb 18;12(2):517. doi: 10.3390/antiox12020517.

An expanded access protocol of RT001 in amyotrophic lateral sclerosis-Initial experience with a lipid peroxidation inhibitor.

Muscle Nerve. 2022 Oct;66(4):421-425. doi: 10.1002/mus.27672. Epub 2022 Jul 29.

Skeletal Muscle-Restricted Expression of Human SOD1 in Transgenic Mice Causes a Fatal ALS-Like Syndrome.

Front Neurol. 2020 Dec 14;11:592851. doi: 10.3389/fneur.2020.592851. eCollection 2020.