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Role of dopamine autoxidation, hydroxyl radical generation, and calcium overload in underlying mechanisms involved in MPTP-induced parkinsonism.

作者信息

Chiueh C C, Miyake H, Peng M T

机构信息

National Institute of Mental Health, National Institutes of Health, Bethesda, Maryland 20892.

出版信息

Adv Neurol. 1993;60:251-8.

PMID:8380519
Abstract
摘要

相似文献

1
Role of dopamine autoxidation, hydroxyl radical generation, and calcium overload in underlying mechanisms involved in MPTP-induced parkinsonism.多巴胺自氧化、羟基自由基生成及钙超载在MPTP诱导的帕金森病潜在机制中的作用。
Adv Neurol. 1993;60:251-8.
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Metabolic aspects of the behavior of MPTP and some analogues.
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3
Biochemical mechanisms underlying MPTP-induced and idiopathic parkinsonism. New vistas.
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[Experimental model of Parkinson disease: mechanisms and anatomo- pathological characteristics of MPTP neurotoxicity].
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Early-stage loss of dopamine uptake-site binding in MPTP-treated monkeys.MPTP 处理的猴子中多巴胺摄取位点结合的早期丧失。
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6
D-deprenyl protects nigrostriatal neurons against 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced dopaminergic neurotoxicity.D-司来吉兰可保护黑质纹状体神经元免受1-甲基-4-苯基-1,2,3,6-四氢吡啶诱导的多巴胺能神经毒性。
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Transferrin receptor regulation in Parkinson's disease and MPTP-treated mice.帕金森病及MPTP处理小鼠中运铁蛋白受体的调控
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Ro 16-6491: a new reversible and highly selective MAO-B inhibitor protects mice from the dopaminergic neurotoxicity of MPTP.Ro 16-6491：一种新型可逆性且高选择性的单胺氧化酶B抑制剂可保护小鼠免受1-甲基-4-苯基-1,2,3,6-四氢吡啶（MPTP）的多巴胺能神经毒性。
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Changes of tyrosine hydroxylase in parkinsonian brains and in the brains of MPTP-treated mice.帕金森病患者大脑及经MPTP处理的小鼠大脑中酪氨酸羟化酶的变化。
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Degeneration of dopaminergic neurons and free radicals. Possible participation of levodopa.
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