Hashimoto K, Kida Y, Takeuchi Y
Division of Neonatology, Matsudo City Hospital, Chiba.
No To Hattatsu. 1993 Sep;25(5):412-6.
We studied periventricular echodensities (PVE) in 154 premature infants under 33 weeks of gestational age. PVEs were classified into four groups due to Pidcock's description, and its relation with periventricular leukomalacia (PVL) was examined as a cause of cerebral palsy. Twelve (11%) of 112 infants with PVE 2 and all of 10 infants with PVE 3 were developed cystic PVL. PVE 3 was therefore considered to be the same as the pathological PVL. Infants of PVE 2 - 3 groups were more premature and lower in birth weight, and they were more often complicated with respiratory distress syndrome, asphyxia, recurrent apneic episode, patent ductus arteriosus and hypocarbia than those of PVE 0 - 1 groups. PVE 2 disappeared in patients with PVL later than in those without PVL.
我们研究了154例孕周小于33周的早产儿的脑室周围回声增强（PVE）情况。根据Pidcock的描述，PVE被分为四组，并研究了其作为脑性瘫痪病因与脑室周围白质软化（PVL）的关系。112例PVE 2级婴儿中有12例（11%）以及10例PVE 3级婴儿全部发展为囊性PVL。因此，PVE 3级被认为与病理性PVL相同。PVE 2 - 3级组的婴儿早产程度更高、出生体重更低，与PVE 0 - 1级组的婴儿相比，他们更常并发呼吸窘迫综合征、窒息、反复呼吸暂停发作、动脉导管未闭和低碳酸血症。PVL患者的PVE 2级消失时间比无PVL患者更晚。
