发育期和成年大鼠长期摄入无机砷后，脑内生物胺、谷氨酸、γ-氨基丁酸水平及谷氨酸脱羧酶活性的区域变化。 - Suppr | 超能文献

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Regional alterations in the levels of brain biogenic amines, glutamate, GABA, and GAD activity due to chronic consumption of inorganic arsenic in developing and adult rats.发育期和成年大鼠长期摄入无机砷后，脑内生物胺、谷氨酸、γ-氨基丁酸水平及谷氨酸脱羧酶活性的区域变化。

Bull Environ Contam Toxicol. 1993 Jan;50(1):100-7. doi: 10.1007/BF00196547.

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Regional alterations of brain biogenic amines and GABA/glutamate levels in rats following chronic lead exposure during neonatal development.新生期发育期间慢性铅暴露后大鼠脑内生物胺及GABA/谷氨酸水平的区域变化

Arch Toxicol. 1990;64(4):305-14. doi: 10.1007/BF01972991.

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Effects of sodium dipropylacetate on gamma-aminobutyric acid and biogenic amines in rat brain.双丙乙酸钠对大鼠脑内γ-氨基丁酸和生物胺的影响。

Chem Pharm Bull (Tokyo). 1977 Sep;25(9):2257-62. doi: 10.1248/cpb.25.2257.

4

Neurochemical changes following the administration of depleters of biogenic monoamines.生物源性单胺耗竭剂给药后的神经化学变化。

Life Sci. 1976 Dec 1;19(11):1663-7. doi: 10.1016/0024-3205(76)90071-0.

5

Effects of benzene vapour on the gamma-aminobutyric acid (GABA) system in rat brain.苯蒸气对大鼠脑内γ-氨基丁酸（GABA）系统的影响。

Biochem Pharmacol. 1975 Nov 15;24(22):2083-7. doi: 10.1016/0006-2952(75)90106-9.

6

Acute and continuous morphine administration on the gamma-aminobutyric acid system in the mouse.急性和持续给予吗啡对小鼠γ-氨基丁酸系统的影响

Prog Neuropsychopharmacol. 1978;2(1):55-64. doi: 10.1016/0364-7722(78)90022-x.

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Effects of amino-oxyacetic acid, ethanolamine-O-sulphate and GABA on the contents of GABA and various amines in brain slices.氨基氧乙酸、乙醇胺 -O -硫酸盐和γ-氨基丁酸对脑切片中γ-氨基丁酸及各种胺类含量的影响。

J Neurochem. 1975 Nov;25(5):573-7. doi: 10.1111/j.1471-4159.1975.tb04370.x.

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Fine distribution of gamma-aminobutyric acid, glutamic acid decarboxylase, and glutamic acid in the rabbit cerebellum.

Exp Neurol. 1988 Mar;99(3):778-83. doi: 10.1016/0014-4886(88)90193-8.

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Brain 3-hydroxybutyrate, glutamate, and GABA in a rat model of dietary obesity.饮食性肥胖大鼠模型中的脑3-羟基丁酸、谷氨酸和γ-氨基丁酸

Physiol Behav. 1989 Mar;45(3):571-7. doi: 10.1016/0031-9384(89)90075-9.

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A convulsant, 3-mercaptopropionic acid, decreases the level of GABA in pancreatic islets of rat as well as that of brain.

Life Sci. 1994;54(12):769-73. doi: 10.1016/0024-3205(94)00445-5.

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Mechanisms Associated with Cognitive and Behavioral Impairment Induced by Arsenic Exposure.砷暴露致认知和行为损伤的相关机制。

Cells. 2023 Oct 28;12(21):2537. doi: 10.3390/cells12212537.

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Metabolomic changes associated with chronic arsenic exposure in a Bangladeshi population.与孟加拉国人群慢性砷暴露相关的代谢组学变化。

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Molecular Mechanism of Arsenic-Induced Neurotoxicity including Neuronal Dysfunctions.砷诱导的神经毒性的分子机制，包括神经元功能障碍。

本文引用的文献

1

The effects of acute and subacute sodium arsenite administration on carbohydrate metabolism.急性和亚急性给予亚砷酸钠对碳水化合物代谢的影响。

Toxicol Appl Pharmacol. 1980 Aug;55(1):126-30. doi: 10.1016/0041-008x(80)90228-8.

2

Arsenic distribution and neurochemical effects in peroral sodium arsenite exposure of rats.大鼠经口亚砷酸钠暴露后的砷分布及神经化学效应

Bull Environ Contam Toxicol. 1983 Mar;30(3):303-8. doi: 10.1007/BF01610137.

3

Acute encephalopathy due to occupational exposure to arsenic.职业性接触砷导致的急性脑病。