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Hydrostatic pulmonary oedema is not a stimulus for prostaglandin synthesis in isolated, perfused lungs [proceedings].

作者信息

Scott E, Vaage J, Wiberg T, Ungar A

出版信息

Br J Pharmacol. 1977 Mar;59(3):445P-446P.

PMID:843694
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1667987/
Abstract
摘要

相似文献

1
Hydrostatic pulmonary oedema is not a stimulus for prostaglandin synthesis in isolated, perfused lungs [proceedings].流体静力性肺水肿并非离体灌注肺中前列腺素合成的刺激因素[会议论文集]。
Br J Pharmacol. 1977 Mar;59(3):445P-446P.
2
Lack of release of prostaglandins from isolated perfused lungs during pulmonary hypertension and oedema.在肺动脉高压和肺水肿期间，离体灌注肺中前列腺素释放缺乏。
Br J Pharmacol. 1979 Feb;65(2):197-204. doi: 10.1111/j.1476-5381.1979.tb07819.x.
3
The effects of graded administration of positive end expiratory pressure on the fluid filtration rate in isolated rabbit lungs, using normal lungs, hydrostatic oedema lungs and oleic acid induced oedema.使用正常肺、静水压性肺水肿肺和油酸诱导的水肿，研究呼气末正压分级给药对离体兔肺液体滤过率的影响。
Intensive Care Med. 1990;16(2):89-94.
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Fiziol Zh (1978). 1989 Jul-Aug;35(4):100-2.
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[Effect of changes in airway pressure and the inspiratory volume on the fluid filtration rate and pulmonary artery pressure in isolated rabbit lungs perfused with blood and acellular solution].[气道压力和吸气量变化对灌注血液和无细胞溶液的离体兔肺液体滤过率和肺动脉压的影响]
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[Mechanism of the development of edema in perfusion of the isolated lungs].[离体肺灌注中水肿形成的机制]
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[Effects of obsidan on the development of pulmonary edema and hemodynamics of the lesser circulation after administration of noradrenaline].[黑曜石对去甲肾上腺素给药后肺水肿发展及微循环血流动力学的影响]
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Negative-feedback loop attenuates hydrostatic lung edema via a cGMP-dependent regulation of transient receptor potential vanilloid 4.负反馈回路通过环磷酸鸟苷（cGMP）依赖性调节瞬时受体电位香草酸亚型4（TRPV4）来减轻静水压性肺水肿。
Circ Res. 2008 Apr 25;102(8):966-74. doi: 10.1161/CIRCRESAHA.107.168724. Epub 2008 Mar 6.
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Activation of rho is involved in the mechanism of hydrogen-peroxide-induced lung edema in isolated perfused rabbit lung.Rho的激活参与了过氧化氢诱导的离体灌注兔肺肺水肿的机制。
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Pulmonary blood flow. A potential factor in the pathogenesis of pulmonary edema.肺血流量。肺水肿发病机制中的一个潜在因素。
J Thorac Cardiovasc Surg. 1984 Jan;87(1):130-5.

引用本文的文献

1
Lack of release of prostaglandins from isolated perfused lungs during pulmonary hypertension and oedema.在肺动脉高压和肺水肿期间，离体灌注肺中前列腺素释放缺乏。
Br J Pharmacol. 1979 Feb;65(2):197-204. doi: 10.1111/j.1476-5381.1979.tb07819.x.

本文引用的文献

1
Vasoconstriction in isolated blood-perfused rabbit lungs and its inhibition by cresols.离体血液灌注兔肺中的血管收缩及其被甲酚抑制的情况
Acta Physiol Scand. 1966 Jan-Feb;66(1):226-40. doi: 10.1111/j.1748-1716.1966.tb03189.x.
2
The release and fate of vaso-active hormones in the circulation.血管活性激素在循环中的释放与转归
Br J Pharmacol. 1969 Feb;35(2):209-42. doi: 10.1111/j.1476-5381.1969.tb07982.x.
3
The release of prostaglandins from lung and other tissues.前列腺素从肺和其他组织中的释放。
Ann N Y Acad Sci. 1971 Apr 30;180:363-85. doi: 10.1111/j.1749-6632.1971.tb53205.x.