Hydrostatic pulmonary oedema is not a stimulus for prostaglandin synthesis in isolated, perfused lungs [proceedings].
作者信息
Scott E, Vaage J, Wiberg T, Ungar A
出版信息
Br J Pharmacol. 1977 Mar;59(3):445P-446P.
Abstract
摘要
相似文献
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Hydrostatic pulmonary oedema is not a stimulus for prostaglandin synthesis in isolated, perfused lungs [proceedings].流体静力性肺水肿并非离体灌注肺中前列腺素合成的刺激因素[会议论文集]。
Br J Pharmacol. 1977 Mar;59(3):445P-446P.
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Negative-feedback loop attenuates hydrostatic lung edema via a cGMP-dependent regulation of transient receptor potential vanilloid 4.负反馈回路通过环磷酸鸟苷（cGMP）依赖性调节瞬时受体电位香草酸亚型4（TRPV4）来减轻静水压性肺水肿。
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Activation of rho is involved in the mechanism of hydrogen-peroxide-induced lung edema in isolated perfused rabbit lung.Rho的激活参与了过氧化氢诱导的离体灌注兔肺肺水肿的机制。
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Pulmonary blood flow. A potential factor in the pathogenesis of pulmonary edema.肺血流量。肺水肿发病机制中的一个潜在因素。
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引用本文的文献
1
Lack of release of prostaglandins from isolated perfused lungs during pulmonary hypertension and oedema.在肺动脉高压和肺水肿期间，离体灌注肺中前列腺素释放缺乏。
Br J Pharmacol. 1979 Feb;65(2):197-204. doi: 10.1111/j.1476-5381.1979.tb07819.x.
本文引用的文献
1
Vasoconstriction in isolated blood-perfused rabbit lungs and its inhibition by cresols.离体血液灌注兔肺中的血管收缩及其被甲酚抑制的情况
Acta Physiol Scand. 1966 Jan-Feb;66(1):226-40. doi: 10.1111/j.1748-1716.1966.tb03189.x.
2
The release and fate of vaso-active hormones in the circulation.血管活性激素在循环中的释放与转归
Br J Pharmacol. 1969 Feb;35(2):209-42. doi: 10.1111/j.1476-5381.1969.tb07982.x.
3
The release of prostaglandins from lung and other tissues.前列腺素从肺和其他组织中的释放。
Ann N Y Acad Sci. 1971 Apr 30;180:363-85. doi: 10.1111/j.1749-6632.1971.tb53205.x.
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