热休克蛋白与缺血性心脏。一种内源性保护机制。 - Suppr

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超能文献

Heat shock proteins and the ischemic heart. An endogenous protective mechanism.

作者信息

Black S C, Lucchesi B R

出版信息

Circulation. 1993 Mar;87(3):1048-51. doi: 10.1161/01.cir.87.3.1048.

DOI:10.1161/01.cir.87.3.1048

PMID:8443885

Abstract

摘要

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Heat shock proteins and the ischemic heart. An endogenous protective mechanism.热休克蛋白与缺血性心脏。一种内源性保护机制。

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Heat shock proteins protect against ischemia and inflammation through multiple mechanisms.热休克蛋白通过多种机制抵御缺血和炎症。

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Presence of heat shock protein 72 in cardiomyocytes after heat stress.

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Small heat shock proteins and protection against ischemic injury in cardiac myocytes.小热休克蛋白与心肌细胞缺血性损伤的保护作用

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引用本文的文献

In vivo gene transfection with heat shock protein 70 enhances myocardial tolerance to ischemia-reperfusion injury in rat.热休克蛋白70体内基因转染增强大鼠心肌对缺血-再灌注损伤的耐受性。

J Clin Invest. 1997 Apr 1;99(7):1645-50. doi: 10.1172/JCI119327.

Stress proteins: a future role in cardioprotection?

Heart. 1996 Aug;76(2):97-8. doi: 10.1136/hrt.76.2.97.

Cardioprotective effects of 70-kDa heat shock protein in transgenic mice.70 kDa热休克蛋白在转基因小鼠中的心脏保护作用。

Proc Natl Acad Sci U S A. 1996 Mar 19;93(6):2339-42. doi: 10.1073/pnas.93.6.2339.

Hsp70 in myocardial ischaemia.心肌缺血中的热休克蛋白70

Experientia. 1994 Nov 30;50(11-12):1075-84. doi: 10.1007/BF01923464.

Overexpression of the rat inducible 70-kD heat stress protein in a transgenic mouse increases the resistance of the heart to ischemic injury.大鼠诱导型70kD热应激蛋白在转基因小鼠中的过表达增强了心脏对缺血性损伤的抵抗力。

J Clin Invest. 1995 Apr;95(4):1446-56. doi: 10.1172/JCI117815.

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