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Stress proteins: a future role in cardioprotection?
Heart. 1996 Aug;76(2):97-8. doi: 10.1136/hrt.76.2.97.
2
[The role of stress proteins HSP70 and the adrenergic system in different resistance to myocardial infarction of August and Wistar genetic rat strains].[应激蛋白HSP70和肾上腺素能系统在八月龄大鼠和Wistar遗传大鼠品系对心肌梗死不同抵抗力中的作用]
Ross Fiziol Zh Im I M Sechenova. 2001 Sep;87(9):1171-7.
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[The significance of the changes in expressions of heat shock protein 70 and heme oxygenase-1 in the myocardium in patients died suddenly due to acute myocardial infarction].[热休克蛋白70和血红素加氧酶-1在急性心肌梗死猝死患者心肌中表达变化的意义]
Zhongguo Wei Zhong Bing Ji Jiu Yi Xue. 2010 Feb;22(2):93-6.
4
[Animal experimental studies on the myocardial metabolism in attacks of angina pectoris and myocardial infarct].
Bibl Cardiol. 1969;22:1-93.
5
[Lactate metabolism in the post-pacing period: prognostic significance in angina pectoris].[起搏后阶段的乳酸代谢：对心绞痛的预后意义]
Boll Soc Ital Cardiol. 1980;25(6):607-10.
6
[From research of cellular stress to various clinical break through innovations?].从细胞应激研究到各种临床突破性创新？
Duodecim. 2001;117(22):2266-72.
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[Metabolic pool of purine and pyrimidine compounds in the venous blood of patients with myocardial infarction and stenocardia].[心肌梗死和心绞痛患者静脉血中嘌呤和嘧啶化合物的代谢池]
Kardiologiia. 1989 Feb;29(2):57-60.
8
[A comparative analysis of the polypeptide composition of heat shock proteins 70 synthesized in Wistar and August rat myocardium].
Biull Eksp Biol Med. 1995 Jul;120(7):30-2.
9
[Disorders of myocardial metabolism in coronary heart disease].[冠心病中的心肌代谢紊乱]
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引用本文的文献

1
Profiling of cell stress protein expression in cardiac tissue of cardiosurgical patients undergoing remote ischemic preconditioning: implications for thioredoxin in cardioprotection.接受远程缺血预处理的心脏手术患者心脏组织中细胞应激蛋白表达的分析：硫氧还蛋白在心脏保护中的意义。
J Transl Med. 2015 Jan 27;13:34. doi: 10.1186/s12967-015-0403-6.

本文引用的文献

1
Specific induction of the 70-kD heat stress proteins by the tyrosine kinase inhibitor herbimycin-A protects rat neonatal cardiomyocytes. A new pharmacological route to stress protein expression?酪氨酸激酶抑制剂赫比霉素-A对70-kD热应激蛋白的特异性诱导可保护大鼠新生心肌细胞。这是应激蛋白表达的一条新的药理学途径吗？
J Clin Invest. 1996 Feb 1;97(3):706-12. doi: 10.1172/JCI118468.
2
Preconditioning: a balanced perspective.预处理：一个平衡的视角。
Br Heart J. 1995 Dec;74(6):575-7. doi: 10.1136/hrt.74.6.575.
3
Differential cytoprotection against heat stress or hypoxia following expression of specific stress protein genes in myogenic cells.在肌原细胞中表达特定应激蛋白基因后对热应激或缺氧的差异性细胞保护作用。
J Mol Cell Cardiol. 1995 Aug;27(8):1669-78. doi: 10.1016/s0022-2828(95)90722-x.
4
Heat shock proteins and the ischemic heart. An endogenous protective mechanism.热休克蛋白与缺血性心脏。一种内源性保护机制。
Circulation. 1993 Mar;87(3):1048-51. doi: 10.1161/01.cir.87.3.1048.
5
Sublethal ischemia alters myocardial antioxidant activity in canine heart.亚致死性缺血改变犬心脏的心肌抗氧化活性。
Am J Physiol. 1993 Jan;264(1 Pt 2):H33-9. doi: 10.1152/ajpheart.1993.264.1.H33.
6
Cardiac stress protein elevation 24 hours after brief ischemia or heat stress is associated with resistance to myocardial infarction.短暂缺血或热应激24小时后心脏应激蛋白升高与心肌梗死抗性相关。
Circulation. 1993 Sep;88(3):1264-72. doi: 10.1161/01.cir.88.3.1264.
7
Clinical implications of the stress response.应激反应的临床意义。
J Clin Invest. 1995 Jan;95(1):3-12. doi: 10.1172/JCI117655.
8
Previous angina alters in-hospital outcome in TIMI 4. A clinical correlate to preconditioning?既往心绞痛对TIMI 4研究中的院内结局有影响。这是一种与预处理相关的临床关联吗？
Circulation. 1995 Jan 1;91(1):37-45. doi: 10.1161/01.cir.91.1.37.
9
Muscular and cardiac adenosine-induced pain is mediated by A1 receptors.肌肉和心脏中腺苷诱导的疼痛是由A1受体介导的。
J Am Coll Cardiol. 1995 Jan;25(1):251-7. doi: 10.1016/0735-1097(94)00352-q.
10
Heat shock proteins and protection against myocardial ischemia.
J Mol Cell Cardiol. 1995 Jan;27(1):45-52. doi: 10.1016/s0022-2828(08)80006-5.

Stress proteins: a future role in cardioprotection?

作者信息

Morris S D, Yellon D M, Marber M S

出版信息

Heart. 1996 Aug;76(2):97-8. doi: 10.1136/hrt.76.2.97.

DOI:10.1136/hrt.76.2.97
PMID:8795464
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC484449/
Abstract
摘要