钴胺素神经病的发病机制：神经系统S-腺苷甲硫氨酸缺乏？
Pathogenesis of cobalamin neuropathy: deficiency of nervous system S-adenosylmethionine?
作者信息
Metz J
机构信息
University of the Witwatersrand, Johannesburg, South Africa.
出版信息
Nutr Rev. 1993 Jan;51(1):12-5. doi: 10.1111/j.1753-4887.1993.tb03046.x.
Abstract
Low levels of cerebrospinal S-adenosylmethionine in association with abnormal myelination in inherited disorders affecting cobalamin and folate metabolism provide new data on a possible mechanism of cobalamin neuropathy.
摘要
在影响钴胺素和叶酸代谢的遗传性疾病中，脑脊液中低水平的S-腺苷甲硫氨酸与髓鞘形成异常相关，这为钴胺素神经病变的可能机制提供了新的数据。
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