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Hormones in the coupling of bone resorption and formation.

作者信息

Martin T J

机构信息

St Vincent's Institute of Medical Research, Fitzroy, Victoria, Australia.

出版信息

Osteoporos Int. 1993;3 Suppl 1:121-5. doi: 10.1007/BF01621884.

DOI:10.1007/BF01621884
PMID:8461537
Abstract
摘要

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本文引用的文献

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The cellular basis of bone resorption.骨吸收的细胞基础。
Clin Orthop Relat Res. 1980 Sep(151):283-93.
2
Parathyroid hormone stimulates bone formation and resorption in organ culture: evidence for a coupling mechanism.甲状旁腺激素在器官培养中刺激骨形成和骨吸收：一种偶联机制的证据。
Proc Natl Acad Sci U S A. 1981 May;78(5):3204-8. doi: 10.1073/pnas.78.5.3204.
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Stimulation of plasminogen activator in osteoblast-like cells by bone-resorbing hormones.
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给予高剂量巨噬细胞集落刺激因子会增加骨转换和骨小梁体积分数。
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Advances in osteoclast biology resulting from the study of osteopetrotic mutations.对骨石化症突变的研究带来破骨细胞生物学的进展。
Hum Genet. 2009 Jan;124(6):561-77. doi: 10.1007/s00439-008-0583-8. Epub 2008 Nov 6.
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Bone turnover and bone collagen maturation in osteoporosis: effects of antiresorptive therapies.骨质疏松症中的骨转换和骨胶原成熟：抗吸收治疗的效果
Osteoporos Int. 2008 Mar;19(3):339-48. doi: 10.1007/s00198-007-0462-5. Epub 2007 Sep 11.
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Assessment of osteoclast number and function: application in the development of new and improved treatment modalities for bone diseases.破骨细胞数量与功能的评估：在骨疾病新型及改良治疗方式研发中的应用
Osteoporos Int. 2007 May;18(5):681-5. doi: 10.1007/s00198-006-0286-8. Epub 2006 Nov 24.
7
Optimising antiresorptive therapies in postmenopausal women: why do we need to give due consideration to the degree of suppression?优化绝经后女性的抗吸收治疗：为何我们需要充分考虑抑制程度？
Drugs. 2006;66(15):1909-18. doi: 10.2165/00003495-200666150-00002.
8
Central IL-1 receptor signaling regulates bone growth and mass.中枢白细胞介素-1受体信号传导调节骨骼生长和骨量。
Proc Natl Acad Sci U S A. 2005 Sep 6;102(36):12956-61. doi: 10.1073/pnas.0502562102. Epub 2005 Aug 26.
9
Acidification of the osteoclastic resorption compartment provides insight into the coupling of bone formation to bone resorption.破骨细胞吸收腔的酸化为骨形成与骨吸收的耦联提供了见解。
Am J Pathol. 2005 Feb;166(2):467-76. doi: 10.1016/S0002-9440(10)62269-9.
4
Inhibition of osteoclastic motility by prostaglandins I2, E1, E2 and 6-oxo-E1.前列环素I2、前列腺素E1、前列腺素E2和6-氧代-前列腺素E1对破骨细胞运动的抑制作用。
J Pathol. 1983 Mar;139(3):383-97. doi: 10.1002/path.1711390313.
5
Role of osteoblasts in hormonal control of bone resorption--a hypothesis.成骨细胞在骨吸收激素调控中的作用——一种假说。
Calcif Tissue Int. 1981;33(4):349-51. doi: 10.1007/BF02409454.
6
Bone cells predispose bone surfaces to resorption by exposure of mineral to osteoclastic contact.骨细胞通过使矿物质暴露于破骨细胞接触而使骨表面易于被吸收。
J Cell Sci. 1985 Jun;76:155-65. doi: 10.1242/jcs.76.1.155.
7
Tumor necrosis factors alpha and beta induce osteoblastic cells to stimulate osteoclastic bone resorption.肿瘤坏死因子α和β可诱导成骨细胞刺激破骨细胞的骨吸收。
J Immunol. 1987 Feb 1;138(3):775-9.
8
Growth factors in bone matrix. Isolation of multiple types by affinity chromatography on heparin-Sepharose.骨基质中的生长因子。通过肝素-琼脂糖亲和层析分离多种类型。
J Biol Chem. 1986 Sep 25;261(27):12665-74.
9
Identification and spatial distribution of retinoids in the developing chick limb bud.发育中鸡胚肢芽中视黄酸的鉴定及空间分布
Nature. 1987;327(6123):625-8. doi: 10.1038/327625a0.
10
Osteoblasts mediate interleukin 1 stimulation of bone resorption by rat osteoclasts.成骨细胞介导白细胞介素1对大鼠破骨细胞骨吸收的刺激作用。
J Exp Med. 1986 Jul 1;164(1):104-12. doi: 10.1084/jem.164.1.104.