Folkers K, Brown R, Judy W V, Morita M
University of Texas, Austin.
Biochem Biophys Res Commun. 1993 Apr 15;192(1):241-5. doi: 10.1006/bbrc.1993.1405.
Over ca. 25 years, assays in animal models established the hematopoietic activities of coenzyme Q's in rhesus monkeys, rabbits, poultry, and children having kwashiorkor. Surprisingly, a virus was found to cause a deficiency of CoQ9. Patients with AIDS showed a-"striking"-clinical response to therapy with CoQ10. The macrophage potentiating activity of CoQ10 was recorded by the carbon clearance method. CoQ10 significantly increased the levels of IgG in patients. Eight new case histories of cancer patients plus two reported cases support the statement that therapy of cancer patients with CoQ10, which has no significant side effect, has allowed survival on an exploratory basis for periods of 5-15 years. These results now justify systematic protocols.
在约25年的时间里，动物模型试验证实了辅酶Q在恒河猴、兔子、家禽以及患有夸希奥科病的儿童体内的造血活性。令人惊讶的是，发现一种病毒会导致辅酶Q9缺乏。艾滋病患者对辅酶Q10治疗呈现出“显著的”临床反应。通过碳清除法记录了辅酶Q10的巨噬细胞增强活性。辅酶Q10显著提高了患者体内的IgG水平。8例癌症患者的新病例记录加上2例已报道病例支持了这一说法，即对癌症患者使用无明显副作用的辅酶Q10进行治疗，已使患者在探索性基础上存活了5至15年。这些结果现在证明了系统方案的合理性。
