Zolli C L
Wills Eye Hospital, Philadelphia, PA 19107, USA.
Ophthalmic Surg. 1995 Jul-Aug;26(4):362-6.
This article presents a novel hypothesis on what causes an involutional entropion. The theory herein proposes that the pathophysiology of entropion is that of an idiopathic dystonia with the locus of dysfunction posed in the rostral brain stem and with stimuli for its causation mediated through the seventh cranial nerve--the facial nerve--by a cord of fibers making its path to the upper, ie, temporooculo-zygomatic ramus of the nerve. In a word, involutional entropion is but a variant of essential blepharospasm and one of the clinical entities within the oculo-oro-facial-cervical family of dystonia disorders.
本文提出了一个关于导致退行性睑内翻病因的新假说。本文中的理论认为，睑内翻的病理生理学是一种特发性肌张力障碍，功能障碍部位位于脑桥上部，其病因是通过第七颅神经（面神经）的一束纤维介导的刺激，该束纤维通向神经的上部，即颞眼颧支。简而言之，退行性睑内翻只是原发性睑痉挛的一种变体，是眼口面部颈部肌张力障碍疾病家族中的临床实体之一。
