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Free radicals and disruption of neuronal ion homeostasis in AD: a role for amyloid beta-peptide?

作者信息

Mattson M P

机构信息

Sanders-Brown Research Center on Aging, University of Kentucky, Lexington 40536-0230, USA.

出版信息

Neurobiol Aging. 1995 Jul-Aug;16(4):679-82; discussion 683. doi: 10.1016/0197-4580(95)00063-k.

DOI:10.1016/0197-4580(95)00063-k
PMID:8544920
Abstract
摘要

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Free radicals and disruption of neuronal ion homeostasis in AD: a role for amyloid beta-peptide?
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[beta-Amyloid, cholinergic neurons and Alzheimer's disease].[β-淀粉样蛋白、胆碱能神经元与阿尔茨海默病]
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Amyloid beta peptide 1-42 disturbs intracellular calcium homeostasis through activation of GluN2B-containing N-methyl-d-aspartate receptors in cortical cultures.淀粉样β肽 1-42 通过激活皮质培养物中含有 GluN2B 的 N-甲基-D-天冬氨酸受体来扰乱细胞内钙稳态。
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