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Cellular cofactors for amyloid beta-peptide-induced cell stress. Moving from cell culture to in vivo.

作者信息

Yan S D, Roher A, Schmidt A M, Stern D M

机构信息

Department of Pathology, Columbia University, New York, New York, USA.

出版信息

Am J Pathol. 1999 Nov;155(5):1403-11. doi: 10.1016/s0002-9440(10)65452-1.

DOI:10.1016/s0002-9440(10)65452-1
PMID:10550293
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1866992/
Abstract
摘要

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Is amyloid causally involved in pathophysiology of Alzheimer's disease?淀粉样蛋白是否在阿尔茨海默病的病理生理学中起因果作用？
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本文引用的文献

1
Elimination of the class A scavenger receptor does not affect amyloid plaque formation or neurodegeneration in transgenic mice expressing human amyloid protein precursors.消除A类清道夫受体不会影响表达人淀粉样蛋白前体的转基因小鼠中的淀粉样斑块形成或神经退行性变。
Am J Pathol. 1999 Nov;155(5):1741-7. doi: 10.1016/S0002-9440(10)65489-2.
2
N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression.蛋白质的N-ε-（羧甲基）赖氨酸加合物是晚期糖基化终产物受体的配体，该受体可激活细胞信号通路并调节基因表达。
J Biol Chem. 1999 Oct 29;274(44):31740-9. doi: 10.1074/jbc.274.44.31740.
3
Dissociation of atherogenesis from aortic accumulation of lipid hydro(pero)xides in Watanabe heritable hyperlipidemic rabbits.渡边遗传性高脂血症兔动脉粥样硬化形成与主动脉脂质氢（过）氧化物蓄积的分离
J Clin Invest. 1999 Jul;104(2):213-20. doi: 10.1172/JCI6391.
4
Is lipid peroxidation relevant to atherogenesis?脂质过氧化与动脉粥样硬化的发生有关吗？
J Clin Invest. 1999 Jul;104(2):135-6. doi: 10.1172/JCI7633.
5
Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse.用β-淀粉样蛋白免疫可减轻PDAPP小鼠的阿尔茨海默病样病理变化。
Nature. 1999 Jul 8;400(6740):173-7. doi: 10.1038/22124.
6
Scavenger receptor deficiency leads to more complex atherosclerotic lesions in APOE3Leiden transgenic mice.清道夫受体缺陷导致载脂蛋白E3莱顿转基因小鼠出现更复杂的动脉粥样硬化病变。
Atherosclerosis. 1999 Jun;144(2):315-21. doi: 10.1016/s0021-9150(98)00332-3.
7
RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides.RAGE介导一种新型促炎轴：一种S100/钙粒蛋白多肽的中枢细胞表面受体。
Cell. 1999 Jun 25;97(7):889-901. doi: 10.1016/s0092-8674(00)80801-6.
8
Integration of endoplasmic reticulum signaling in health and disease.内质网信号在健康与疾病中的整合
Nat Med. 1999 Jul;5(7):745-51. doi: 10.1038/10466.
9
Inflammatory pathogenesis in Alzheimer's disease: biological mechanisms and cognitive sequeli.阿尔茨海默病的炎症发病机制：生物学机制与认知后遗症
Neurosci Biobehav Rev. 1999 May;23(5):615-33. doi: 10.1016/s0149-7634(98)00058-x.
10
Translating cell biology into therapeutic advances in Alzheimer's disease.将细胞生物学转化为阿尔茨海默病的治疗进展。
Nature. 1999 Jun 24;399(6738 Suppl):A23-31. doi: 10.1038/399a023.