Suwalski K, Pytkowski M, Zelazny P, Wojciechowski D, Sitkowska E, Sadowski Z, Sitkowski W
Second Department of Cardiosurgery, National Institute of Cardiology, Warsaw, Poland.
Cardiovasc Surg. 1995 Oct;3(5):545-7. doi: 10.1016/0967-2109(95)94456-7.
Fifty patients with drug-resistant, recurrent tachyarrhythmias causing Wolff-Parkinson-White syndrome underwent surgery between 1990 and 1992. All recognized surgical methods for accessory pathway destruction were performed. Epicardial electric shock ablation was first used as a method of surgically destroying an accessory atrioventricular pathway in 1983. This technique avoids the need for cardioplegia and hypothermia during operation. The procedure is based on the application of a series of two to five electrical shocks (50-150 J) to the region of the atrioventricular groove where the accessory pathway has been previously located. Some 32 patients with a left free wall accessory pathway underwent this operation. Cardioplegia and hypothermia were not required in 22 patients with an accessory pathway located in the left lateral position. In the second group comprising ten patients with a left lateral accessory pathway, four were diagnosed as having a second pathway and four had concomitant heart pathology such as coronary artery disease -- one had an atrial septal defect and another had a ventricular septal defect. Accessory pathway ablation was carried out in these ten patients using epicardial electric shock under normothermic cardiopulmonary bypass. Concomitant heart pathology was corrected at the second stage of the operation under cardiopulmonary bypass with cardioplegia and hypothermia. Postoperative electrophysiological studies confirmed that the accessory pathway had been destroyed in all patients. The only side effects of epicardial electric shock application were transient ST elevation < 1 mm in four patients, transient atrioventricular bloc in two and moderate sinus tachycardia in three.(ABSTRACT TRUNCATED AT 250 WORDS)
1990年至1992年间,50例因耐药性复发性快速心律失常导致预激综合征的患者接受了手术。所有公认的用于破坏旁路的手术方法均被采用。1983年,心外膜电击消融术首次被用作手术破坏房室旁路的一种方法。该技术避免了手术期间使用心脏停搏和低温。该手术基于对房室沟区域施加一系列两到五次电击(50 - 150焦耳),该区域先前是旁路所在位置。约32例左游离壁旁路患者接受了该手术。22例位于左侧的旁路患者无需心脏停搏和低温。在第二组10例左侧旁路患者中,4例被诊断有第二条旁路,4例伴有心脏病变,如冠状动脉疾病,1例有房间隔缺损,另1例有室间隔缺损。这10例患者在常温体外循环下用心外膜电击进行旁路消融。在体外循环下心脏停搏和低温的第二阶段手术中纠正了伴随的心脏病变。术后电生理研究证实所有患者的旁路均被破坏。心外膜电击应用的唯一副作用是4例患者出现短暂ST段抬高<1毫米,2例出现短暂房室传导阻滞,3例出现中度窦性心动过速。(摘要截选至250字)