Mortel R, Allegra J C, Demers L M, Harvey H A, Trautlein J, Nahhas W, White D, Gillin M A, Lipton A
Cancer. 1977 May;39(5):2201-3. doi: 10.1002/1097-0142(197705)39:5<2201::aid-cncr2820390538>3.0.co;2-9.
Prostaglandin E produced by tumors has recently been implicated as a mechanism by which tumors may subvert the immune system and grow despite their antigenicity. Arterial and venous determinations of prostaglandin E were performed in eleven patients with gynecologic malignancy. No significant difference was found when arterial and venous levels were compared and there was no difference in venous PGE levels when subjects with cancer were compared to patients with benign gynecologic disease.
肿瘤产生的前列腺素E最近被认为是肿瘤尽管具有抗原性却可能颠覆免疫系统并生长的一种机制。对11例妇科恶性肿瘤患者进行了动脉和静脉前列腺素E的测定。比较动脉和静脉水平时未发现显著差异，将癌症患者与妇科良性疾病患者进行比较时，静脉PGE水平也没有差异。
