正常肾素型原发性高血压中肾素-血管紧张素-醛固酮关系的改变 - Suppr

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Altered renin-angiotensin-aldosterone relationships in normal renin essential hypertension.

作者信息

Moore T J, Williams G H, Dluhy R G, Bavli S Z, Himathongkam T, Greenfield M

出版信息

Circ Res. 1977 Aug;41(2):167-71. doi: 10.1161/01.res.41.2.167.

DOI:10.1161/01.res.41.2.167

PMID:872289

Abstract

摘要

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Altered renin-angiotensin-aldosterone relationships in normal renin essential hypertension.正常肾素型原发性高血压中肾素-血管紧张素-醛固酮关系的改变

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Clinical conference: Essential hypertension: new concepts about mechanisms.临床研讨会：原发性高血压：机制的新概念

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Interruption of the renin-angiotensin system in hypertensive patients by captopril induces sustained reduction in aldosterone secretion, potassium retention and natruiresis.卡托普利对高血压患者肾素 - 血管紧张素系统的阻断可导致醛固酮分泌持续减少、钾潴留和利钠作用。

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Regulation of angiotensin II receptors in the rat adrenal cortex by dietary electrolytes.饮食电解质对大鼠肾上腺皮质中血管紧张素 II 受体的调节作用。

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Low-renin hypertension.

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引用本文的文献

Renin-hyporesponsiveness in essential hypertension. Dissociation between plasma renin and catecholamines or aldosterone following furosemide.

Klin Wochenschr. 1980 May 2;58(9):457-65. doi: 10.1007/BF01476800.

Defect in the sodium-modulated tissue responsiveness to angiotensin II in essential hypertension.原发性高血压中钠调节的组织对血管紧张素II反应性的缺陷。

J Clin Invest. 1983 Dec;72(6):2115-24. doi: 10.1172/JCI111176.

Decreased adrenal responsiveness to angiotensin II: a defect present in spontaneously hypertensive rats. A possible model of human essential hypertension.肾上腺对血管紧张素II反应性降低：自发性高血压大鼠存在的一种缺陷。人类原发性高血压的一种可能模型。

J Clin Invest. 1982 Jan;69(1):31-7. doi: 10.1172/jci110438.

Correction of abnormal renal blood flow response to angiotensin II by converting enzyme inhibition in essential hypertensives.通过血管紧张素转换酶抑制纠正原发性高血压患者对血管紧张素II的异常肾血流反应。

J Clin Invest. 1985 Apr;75(4):1285-90. doi: 10.1172/JCI111828.

Abnormal adrenal responsiveness and angiotensin II dependency in high renin essential hypertension.高肾素性原发性高血压中肾上腺反应异常及对血管紧张素II的依赖性

J Clin Invest. 1979 Nov;64(5):1270-6. doi: 10.1172/JCI109582.

The adrenal receptor for angiotensin II is altered in essential hypertension.原发性高血压患者体内的血管紧张素 II 肾上腺受体发生了改变。

J Clin Invest. 1979 Mar;63(3):419-27. doi: 10.1172/JCI109318.

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