Senichenkova I N, Chebotar' N A
Ontogenez. 1996 Mar-Apr;27(2):108-13.
Iron-deficiency anemia induced in the maternal organism markedly enhances embryotoxic and teratogenic effects of the studied pollutants: gasoline and formaldehyde. The prenatal effects of these drugs against the background of iron-deficiency state in pregnant females leads to development of the main feature of the tissue hypoxia, decompensated metabolic acidosis, in both the maternal organism and 20-day embryos.
母体缺铁性贫血显著增强了所研究污染物（汽油和甲醛）的胚胎毒性和致畸作用。在怀孕雌性动物缺铁状态背景下，这些药物的产前影响会导致母体和20日龄胚胎出现组织缺氧和代偿性代谢性酸中毒的主要特征。
