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细胞对损伤的反应。I. 犬失血性休克所致肝细胞损伤的超微结构与生化研究

Subcellular reactions to injury. I. Ultrastructural and biochemical investigations on the hepatic cellular damage produced by haemorrhagic shock in dogs.

作者信息

Russo M A, Conforti A

出版信息

J Pathol. 1977 Feb;121(2):107-13. doi: 10.1002/path.1711210207.

DOI:10.1002/path.1711210207
PMID:874631
Abstract

The ultrastructural changes produced by haemorrhagic shock in hepatocytes have been studied in dogs. The associated biochemical and biophysical system alterations allow us to propose a unitary pathogenetic mechanism of the cellular injury based on oxygen deprivation and its effect on the function and structure of the cellular compartments.

摘要

对狗肝细胞因失血性休克产生的超微结构变化进行了研究。相关的生化和生物物理系统改变使我们能够基于缺氧及其对细胞区室功能和结构的影响，提出细胞损伤的统一发病机制。

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Subcellular reactions to injury. I. Ultrastructural and biochemical investigations on the hepatic cellular damage produced by haemorrhagic shock in dogs.细胞对损伤的反应。I. 犬失血性休克所致肝细胞损伤的超微结构与生化研究
J Pathol. 1977 Feb;121(2):107-13. doi: 10.1002/path.1711210207.
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[Oxygen supply of the dog liver in situ in experimental hemorrhagic shock].[实验性失血性休克时犬肝脏原位的氧供应]
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Hepatocellular ultrastructure in dogs with hypovolemic shock.低血容量性休克犬的肝细胞超微结构
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