Burney T L, Senapati M, Desai S, Choudhary S T, Badlani G H
Department of Urology, Long Island Jewish Medical Center, New Hyde Park, New York, USA.
J Urol. 1996 Nov;156(5):1748-50. doi: 10.1016/s0022-5347(01)65498-3.
We evaluated the effects of an acute cerebrovascular accident on the lower urinary tract and correlated the site of cerebrovascular accident with findings on urodynamic study.
A total of 45 men and 15 women underwent a complete urodynamic study with electromyography within 72 hours of a cerebrovascular accident. Patients were divided into 5 different groups based on urodynamic findings.
The majority of cortical and internal capsule lesions resulted in detrusor hyperreflexia. A total of 28 patients (47%) had urinary retention, mainly due to detrusor areflexia (75%). Of 20 patients with hemorrhagic infarcts 17 (85%) had areflexia, compared to only 4 of 40 (10%) with ischemic infarcts. All 6 patients with cerebellar infarction had detrusor areflexia.
Our results confirm many previously reported findings. In addition, there was a specific correlation of cerebellar and hemorrhagic infarctions with detrusor areflexia.
我们评估了急性脑血管意外对下尿路的影响，并将脑血管意外的部位与尿动力学研究结果进行关联。
总共45名男性和15名女性在脑血管意外发生后72小时内接受了完整的尿动力学检查及肌电图检查。根据尿动力学检查结果，患者被分为5个不同组。
大多数皮质和内囊病变导致逼尿肌反射亢进。共有28例患者（47%）出现尿潴留，主要原因是逼尿肌无反射（75%）。在20例出血性梗死患者中，17例（85%）有无反射，相比之下，40例缺血性梗死患者中只有4例（10%）有无反射。所有6例小脑梗死患者均有逼尿肌无反射。
我们的结果证实了许多先前报道的发现。此外，小脑梗死和出血性梗死与逼尿肌无反射存在特定关联。
