Helicobacter pylori, gastric acid, and duodenal gastric metaplasia.
作者信息
McColl K E
机构信息
Department of Medicine and Therapeutics, Gardiner Institute, Western Infirmary, Glasgow.
出版信息
Gut. 1996 Oct;39(4):615-6. doi: 10.1136/gut.39.4.615.
Abstract
摘要
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本文引用的文献
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Basal and stimulated gastrin levels and gastric acid output five months after therapy for Helicobacter pylori eradication in duodenal ulcer patients.十二指肠溃疡患者根除幽门螺杆菌治疗五个月后的基础和刺激胃泌素水平及胃酸分泌量
J Clin Gastroenterol. 1996 Mar;22(2):90-5. doi: 10.1097/00004836-199603000-00003.
2
Eradication of Helicobacter pylori in patients with duodenal ulcer lowers basal and peak acid outputs to gastrin releasing peptide and pentagastrin.根除十二指肠溃疡患者的幽门螺杆菌可降低对胃泌素释放肽和五肽胃泌素的基础胃酸分泌量和胃酸分泌峰值。
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Pathogenesis of gastric metaplasia of the human duodenum: role of Helicobacter pylori, gastric acid, and ulceration.人十二指肠胃化生的发病机制：幽门螺杆菌、胃酸及溃疡的作用。
Gastroenterology. 1996 Feb;110(2):452-8. doi: 10.1053/gast.1996.v110.pm8566592.
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Acid secretion and sensitivity to gastrin in patients with duodenal ulcer: effect of eradication of Helicobacter pylori.十二指肠溃疡患者的胃酸分泌及对胃泌素的敏感性：根除幽门螺杆菌的影响
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Behaviour of acid secretion, gastrin release, serum pepsinogen I, and gastric emptying of liquids over six months from eradication of helicobacter pylori in duodenal ulcer patients. A controlled study.十二指肠溃疡患者根除幽门螺杆菌后六个月内胃酸分泌、胃泌素释放、血清胃蛋白酶原I及液体胃排空的行为。一项对照研究。
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