Kalinskaia L N, Kononenko V Ia
Fiziol Zh Im I M Sechenova. 1996 Apr;82(4):60-4.
Administration of angiotensin-converting enzyme inhibitor captopril inhibited the activity of renin-angiotensin system (RAS), increased leukin-enkephalin level in the hypothalamus and adenohypophysis. The data obtained suggest that the enzyme is an important link in the mechanism of interaction between the renin-angiotensin and enkephalinergic systems in rats.
给予血管紧张素转换酶抑制剂卡托普利可抑制肾素-血管紧张素系统(RAS)的活性,增加下丘脑和腺垂体中亮氨酸脑啡肽的水平。所获得的数据表明,该酶是大鼠肾素-血管紧张素系统与脑啡肽能系统相互作用机制中的一个重要环节。