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SCID mice and the study of parasitic disease.

作者信息

Seydel K B, Stanley S L

机构信息

Department of Medicine and Molecular Microbiology, Washington University, St. Louis, Missouri 63110, USA.

出版信息

Clin Microbiol Rev. 1996 Apr;9(2):126-34. doi: 10.1128/CMR.9.2.126.

DOI:10.1128/CMR.9.2.126
PMID:8964031
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC172886/
Abstract
摘要

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SCID mice and the study of parasitic disease.重症联合免疫缺陷小鼠与寄生虫病研究
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2
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4
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5
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Effect of surface antigen-1 (SA-1) immune lymphocyte subsets and naive cell subsets in protecting scid mice from initial and persistent infection with Cryptosporidium parvum.表面抗原-1（SA-1）免疫淋巴细胞亚群和初始细胞亚群在保护重度联合免疫缺陷（scid）小鼠免受微小隐孢子虫初次和持续感染中的作用。
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CD4+ T cell mediated destruction of xenografts within cell-impermeable membranes in the absence of CD8+ T cells and B cells.在缺乏CD8+T细胞和B细胞的情况下，CD4+T细胞介导细胞不可渗透膜内异种移植物的破坏。
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本文引用的文献

1
[Essential lymphocytophthisis; new clinical aspect of infant pathology].[原发性淋巴细胞消耗症；婴儿病理学的新临床方面]
Ann Paediatr. 1950 Jul-Aug;175(1-2):1-32.
2
Human Plasmodium liver stages in SCID mice: a feasible model?严重联合免疫缺陷（SCID）小鼠体内的人疟原虫肝期：一个可行的模型？
Parasitol Today. 1995 May;11(5):169-71. doi: 10.1016/0169-4758(95)80147-2.
3
Expression of major histocompatibility complex class II antigens and levels of interferon-gamma, tumour necrosis factor, and interleukin-6 in cerebrospinal fluid and serum in Toxoplasma gondii-infected SCID and immunocompetent C.B-17 mice.弓形虫感染的重症联合免疫缺陷（SCID）小鼠和免疫健全的C.B-17小鼠脑脊液及血清中主要组织相容性复合体II类抗原的表达以及γ干扰素、肿瘤坏死因子和白细胞介素-6的水平
Immunology. 1993 Mar;78(3):430-5.
4
The fate of human peripheral blood lymphocytes after transplantation into SCID mice.人类外周血淋巴细胞移植到重症联合免疫缺陷小鼠后的命运。
Eur J Immunol. 1993 May;23(5):1023-8. doi: 10.1002/eji.1830230506.
5
Interleukin-2 receptor gamma chain mutation results in X-linked severe combined immunodeficiency in humans.白细胞介素-2受体γ链突变导致人类X连锁重症联合免疫缺陷。
Cell. 1993 Apr 9;73(1):147-57. doi: 10.1016/0092-8674(93)90167-o.
6
Cryptosporidium parvum infection after abrogation of natural killer cell activity in normal and severe combined immunodeficiency mice.正常及严重联合免疫缺陷小鼠自然杀伤细胞活性被消除后隐孢子虫感染情况
J Parasitol. 1993 Apr;79(2):295-7.
7
Reconstitution of C.B-17 scid mice with BALB/c T cells initiates a T helper type-1 response and renders them capable of healing Leishmania major infection.用BALB/c T细胞重建C.B-17重度联合免疫缺陷小鼠会引发1型辅助性T细胞反应，并使它们能够治愈利什曼原虫主要感染。
Eur J Immunol. 1993 Jan;23(1):262-8. doi: 10.1002/eji.1830230141.
8
Theileria sergenti infection in the Bo-RBC-SCID mouse model.
Parasitol Res. 1993;79(6):466-70. doi: 10.1007/BF00931584.
9
Effect of orally administered monoclonal antibody on persistent Cryptosporidium parvum infection in scid mice.口服单克隆抗体对重度联合免疫缺陷小鼠持续性微小隐孢子虫感染的影响。
Infect Immun. 1993 Nov;61(11):4906-8. doi: 10.1128/iai.61.11.4906-4908.1993.
10
The SCID mouse as a laboratory model for development of the exoerythrocytic stages of human and rodent malaria.严重联合免疫缺陷小鼠作为人类和啮齿动物疟疾红细胞外期发育的实验室模型。
Exp Parasitol. 1993 Sep;77(2):257-60. doi: 10.1006/expr.1993.1083.