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[暂时性改善后缺氧性脑损伤的延迟症状]

[Delayed symptoms of hypoxic brain damage after temporary improvement].

作者信息

Tvedt B, Krogstad J M, Berstad J, Roalsøe S

机构信息

Sunnaas sykehus, Nesoddtangen.

出版信息

Tidsskr Nor Laegeforen. 1996 Oct 20;116(25):3009-10.

PMID:8975425
Abstract

A shipyard worker was doing welding work inside a pipe with argon as cover gas. After taking a break he fainted while controlling the weld, also inside the pipe. He was rescued after 15-20 minutes and regained consciousness after a few hours. Two days later he was discharged from hospital in apparently good health. After a week he returned to the hospital suffering from confusion, failing memory, aphasia, apraxia and urinary incontinence. MR showed elevated signal intensity bilaterally in the caudate nucleus. He improved gradually and six months later was given less demanding work. The course of his illness is consistent with delayed neuropsychiatric sequelae after hypoxia, probably due to displacement of oxygen by argon. Delayed symptoms are caused by demyelination in the subcortical white matter.

摘要

一名造船厂工人在以氩气作为保护气体的管道内进行焊接工作。休息片刻后,他在管道内控制焊接时晕倒。15至20分钟后他被救出,数小时后恢复意识。两天后他出院,当时身体状况明显良好。一周后,他因出现意识模糊、记忆力减退、失语、失用症和尿失禁再次入院。磁共振成像显示双侧尾状核信号强度升高。他逐渐康复,六个月后被安排从事要求较低的工作。他的病程符合缺氧后迟发性神经精神后遗症,可能是由于氩气取代了氧气。延迟症状是由皮质下白质脱髓鞘引起的。

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