Pugachev M K
Arkh Anat Gistol Embriol. 1977 May;72(5):73-7.
The problem was studied on albino rats: five rats served as controls; five--were overheated in a thermochamber at 45 degrees C up to the state of motor excitation (the first series); five--up to the heat stroke (the second series). By means of cytometry it was statistically stated that the cross section area of the fascicular zone spongiocytes of the adrenal cortex in the first series decreased by 14,77% in comparison with the control. The cross section area of the adrenocorticocytes of the reticular zone in the animals of the second series decreased by 7,91%. The vascular reaction of the adrenal cortex in the animals of the first series was manifested in hyperemia, which increased in animals of the second series. Hence, the adrenal cortex thickness is the result of blood congestion rather than derived from real hypertrophy; an abrupt vasodilatation disturbs the parallelism in spongiocytes arrangement, that gives a semblance that the thickness of the reticular cortex zone goes at the expense of the fascicular zone.
五只大鼠作为对照；五只在45摄氏度的热室中过热至运动兴奋状态（第一组）；五只过热至中暑状态（第二组）。通过细胞计数法经统计学表明，第一组肾上腺皮质束状带海绵状细胞的横截面积与对照组相比减少了14.77%。第二组动物网状带肾上腺皮质细胞的横截面积减少了7.91%。第一组动物肾上腺皮质的血管反应表现为充血，第二组动物充血加剧。因此，肾上腺皮质厚度是血液充血的结果，而非真正的肥大；突然的血管扩张扰乱了海绵状细胞排列的平行性，这给人一种网状皮质带厚度是以束状带为代价的假象。
