Leĭtes A L, Akylbekov K M, Gabitov V Kh
Arkh Anat Gistol Embriol. 1977 Jul;73(7):21-6.
Modelling of the brain ischemic hypoxia, the hypophysis including, by ligation of the left internal carotid artery in dogs results in weight enlargement of the hypophysis, marked edema of the parenchyma, discomplexity of the cellular elements of the anterior pituitary lobe with morphological signs of hypofunctional condition of the increting cells in the organ studied. Ischemic hypoxia produces lumen augmentation of blood vessels in some areas of the anterior pituitary lobe with resulting increased circulatory capacity in the anterior pituitary lobe. Comparing with the data available in the literature, we conclude that blood circulation manifests a nonspecific reaction to different hypoxic models, while cellular reaction is more specific.
通过结扎犬的左颈内动脉对包括垂体在内的脑缺血缺氧进行建模，结果导致垂体重量增加、实质明显水肿、垂体前叶细胞成分紊乱，且所研究器官中分泌细胞出现功能减退状态的形态学征象。缺血缺氧使垂体前叶某些区域的血管腔增大，从而导致垂体前叶循环能力增强。与文献中的现有数据相比，我们得出结论，血液循环对不同的缺氧模型表现出非特异性反应，而细胞反应则更具特异性。
