Sergeeva E D, Semchenko V V
Anesteziol Reanimatol. 1996 Sep-Oct(5):56-8.
Quantitative analysis of the neuronal populations of the cerebellar cortex was carried out in white rats after reanimation. Clinical death was modeled by 10-min clamping of the neurovascular plexus near the heart. The material for analysis was collected at the end of ischemia, 90 min, 1, 3, and 7 days after it. There are about 8 neurons of the molecular layer and 250 granular cells per pyramidal neuron in an intact cerebellar cortex of white rats. Staring from day 3 of the postreanimation period, the number of granular cells per pyramidal neuron increases, whereas the ratio of the molecular layer neurons and pyramidal neurons differs but negligibly from the control values over the entire follow-up period. Different sensitivity of various neuron populations to an ischemic exposure leads to the formation of the structural basis for the predominance of stimulating pulsation in the cerebellar cortex and reduces its inhibitory effect on the cerebellar nucleus in the postreanimation period.
复苏后对白鼠小脑皮质神经元群体进行了定量分析。通过夹闭心脏附近的神经血管丛10分钟模拟临床死亡。分析材料在缺血结束时、缺血后90分钟、1天、3天和7天采集。在正常的白鼠小脑皮质中，每个锥体神经元约有8个分子层神经元和250个颗粒细胞。从复苏后第3天开始，每个锥体神经元的颗粒细胞数量增加，而在整个随访期间，分子层神经元与锥体神经元的比例与对照值相比差异可忽略不计。不同神经元群体对缺血暴露的敏感性不同，导致在复苏后时期小脑皮质中刺激搏动占优势的结构基础形成，并降低了其对小脑核的抑制作用。
