Multiple pathways for the initiation of T helper 2 (Th2) responses.
作者信息
Coffman R L, von der Weid T
机构信息
Department of Immunology, DNAX Research Institute of Molecular and Cellular Biology, Palo Alto, California 94304, USA.
出版信息
J Exp Med. 1997 Feb 3;185(3):373-5. doi: 10.1084/jem.185.3.373.
Abstract
摘要
相似文献
1
Multiple pathways for the initiation of T helper 2 (Th2) responses.辅助性T细胞2（Th2）应答起始的多种途径。
J Exp Med. 1997 Feb 3;185(3):373-5. doi: 10.1084/jem.185.3.373.
2
Expression of Th2 cytokines and the stable Th2 marker ST2L in the absence of IL-4 during Leishmania major infection.在硕大利什曼原虫感染期间，白细胞介素-4缺乏时Th2细胞因子及稳定的Th2标志物ST2L的表达
Eur J Immunol. 1999 Nov;29(11):3621-8. doi: 10.1002/(SICI)1521-4141(199911)29:11<3621::AID-IMMU3621>3.0.CO;2-Z.
3
IL-4 instructs TH1 responses and resistance to Leishmania major in susceptible BALB/c mice.白细胞介素-4在易感性BALB/c小鼠中指导辅助性T细胞1型反应及对硕大利什曼原虫的抗性。
Nat Immunol. 2001 Nov;2(11):1054-60. doi: 10.1038/ni725.
4
5
A Leishmania major response locus identified by interval-specific congenic mapping of a T helper type 2 cell bias-controlling quantitative trait locus.通过2型辅助性T细胞偏向控制数量性状基因座的区间特异性同源定位鉴定出的一个利什曼原虫主要反应基因座。
J Exp Med. 2004 Dec 20;200(12):1605-12. doi: 10.1084/jem.20040334. Epub 2004 Dec 13.
6
Interleukin-4 (IL-4) and IL-10 collude in vaccine failure for novel exacerbatory antigens in murine Leishmania major infection.白细胞介素-4（IL-4）和白细胞介素-10在小鼠利什曼原虫主要感染中针对新型加重抗原的疫苗失败中相互勾结。
Infect Immun. 2005 Nov;73(11):7620-8. doi: 10.1128/IAI.73.11.7620-7628.2005.
7
Induction of a Th2 population from a polarized Leishmania-specific Th1 population by in vitro culture with IL-4.通过与白细胞介素-4进行体外培养，从极化的利什曼原虫特异性Th1群体诱导出Th2群体。
J Immunol. 1995 Apr 15;154(8):3779-87.
8
9
Organ-specific distribution of CD4+ T1/ST2+ Th2 cells in Leishmania major infection.硕大利什曼原虫感染中CD4+ T1/ST2+ Th2细胞的器官特异性分布。
Eur J Immunol. 2002 Sep;32(9):2450-9. doi: 10.1002/1521-4141(200209)32:9<2450::AID-IMMU2450>3.0.CO;2-O.
10
Interleukin-6 deficiency influences cytokine expression in susceptible BALB mice infected with Leishmania major but does not alter the outcome of disease.白细胞介素-6缺乏影响感染硕大利什曼原虫的易感BALB小鼠的细胞因子表达，但不改变疾病结局。
Infect Immun. 2001 Aug;69(8):5189-92. doi: 10.1128/IAI.69.8.5189-5192.2001.
引用本文的文献
1
A Review on Maternal and Infant Microbiota and Their Implications for the Prevention and Treatment of Allergic Diseases.母婴微生物组及其对预防和治疗过敏性疾病的影响的综述。
Nutrients. 2023 May 26;15(11):2483. doi: 10.3390/nu15112483.
2
Interactions between Innate Lymphoid Cells and Cells of the Innate and Adaptive Immune System.天然淋巴细胞与天然免疫和适应性免疫系统细胞之间的相互作用。
Front Immunol. 2017 Oct 30;8:1422. doi: 10.3389/fimmu.2017.01422. eCollection 2017.
3
Preventive effects of 1,25-(OH)2VD3 against ConA-induced mouse hepatitis through promoting vitamin D receptor gene expression.1,25-(OH)2VD3 通过促进维生素 D 受体基因表达对 ConA 诱导的小鼠肝炎的预防作用。
Acta Pharmacol Sin. 2010 Jun;31(6):703-8. doi: 10.1038/aps.2010.53.
4
Priming for T helper type 2 differentiation by interleukin 2-mediated induction of interleukin 4 receptor alpha-chain expression.白细胞介素2介导白细胞介素4受体α链表达诱导促进2型辅助性T细胞分化
Nat Immunol. 2008 Nov;9(11):1288-96. doi: 10.1038/ni.1656. Epub 2008 Sep 28.
5
Type 2 immunity is controlled by IL-4/IL-13 expression in hematopoietic non-eosinophil cells of the innate immune system.2型免疫由固有免疫系统中造血非嗜酸性粒细胞的IL-4/IL-13表达所调控。
J Exp Med. 2006 Jun 12;203(6):1435-46. doi: 10.1084/jem.20052448. Epub 2006 May 15.
6
Identification of an interleukin (IL)-25-dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion.鉴定一种依赖白细胞介素（IL）-25的细胞群体，该群体在蠕虫排出开始时提供IL-4、IL-5和IL-13。
J Exp Med. 2006 Apr 17;203(4):1105-16. doi: 10.1084/jem.20051615. Epub 2006 Apr 10.
7
Both free-living and parasitic nematodes induce a characteristic Th2 response that is dependent on the presence of intact glycans.自由生活的线虫和寄生线虫都会引发一种典型的Th2反应，这种反应依赖于完整聚糖的存在。
Infect Immun. 2004 Jan;72(1):398-407. doi: 10.1128/IAI.72.1.398-407.2004.
8
Diversity in the oesophageal phenotypic response to gastro-oesophageal reflux: immunological determinants.食管对胃食管反流的表型反应的多样性：免疫决定因素。
Gut. 2002 Apr;50(4):451-9. doi: 10.1136/gut.50.4.451.
9
Immunology of parasitic helminth infections.寄生性蠕虫感染的免疫学
Infect Immun. 2002 Feb;70(2):427-33. doi: 10.1128/IAI.70.2.427-433.2002.
10
Induction by a lactic acid bacterium of a population of CD4(+) T cells with low proliferative capacity that produce transforming growth factor beta and interleukin-10.一种乳酸菌诱导产生一群增殖能力低、能产生转化生长因子β和白细胞介素-10的CD4(+) T细胞。
Clin Diagn Lab Immunol. 2001 Jul;8(4):695-701. doi: 10.1128/CDLI.8.4.695-701.2001.
本文引用的文献
1
Interleukin (IL)-6 directs the differentiation of IL-4-producing CD4+ T cells.白细胞介素（IL）-6指导产生IL-4的CD4+T细胞的分化。
J Exp Med. 1997 Feb 3;185(3):461-9. doi: 10.1084/jem.185.3.461.
2
Schistosoma mansoni egg-induced early IL-4 production is dependent upon IL-5 and eosinophils.曼氏血吸虫卵诱导的早期白细胞介素-4产生依赖于白细胞介素-5和嗜酸性粒细胞。
J Exp Med. 1996 Nov 1;184(5):1871-8. doi: 10.1084/jem.184.5.1871.
3
4
Expansion of natural (NK1+) T cells that express alpha beta T cell receptors in transporters associated with antigen presentation-1 null and thymus leukemia antigen positive mice.在与抗原呈递相关的转运蛋白1缺陷且胸腺白血病抗原阳性的小鼠中，表达αβ T细胞受体的天然（NK1 +）T细胞的扩增。
J Exp Med. 1996 Oct 1;184(4):1579-84. doi: 10.1084/jem.184.4.1579.
5
Beta 2-microglobulin-dependent T cells are dispensable for allergen-induced T helper 2 responses.β2微球蛋白依赖性T细胞对于变应原诱导的辅助性T细胞2型反应并非必需。
J Exp Med. 1996 Oct 1;184(4):1507-12. doi: 10.1084/jem.184.4.1507.
6
Beta 2-microglobulin-dependent NK1.1+ T cells are not essential for T helper cell 2 immune responses.β2-微球蛋白依赖性NK1.1+ T细胞对于辅助性T细胞2型免疫反应并非必不可少。
J Exp Med. 1996 Oct 1;184(4):1295-304. doi: 10.1084/jem.184.4.1295.
7
Increased interleukin 4 and immunoglobulin E production in transgenic mice overexpressing NK1 T cells.过表达NK1 T细胞的转基因小鼠中白细胞介素4和免疫球蛋白E产生增加。
J Exp Med. 1996 Oct 1;184(4):1285-93. doi: 10.1084/jem.184.4.1285.
8
Resistance to Leishmania major induced by tolerance to a single antigen.对单一抗原产生耐受性所诱导的对硕大利什曼原虫的抗性。
Science. 1996 Oct 18;274(5286):421-3. doi: 10.1126/science.274.5286.421.
9
Impaired neutrophil response and CD4+ T helper cell 1 development in interleukin 6-deficient mice infected with Candida albicans.白细胞介素6缺陷型小鼠感染白色念珠菌后中性粒细胞反应受损及CD4 +辅助性T细胞1发育异常
J Exp Med. 1996 Apr 1;183(4):1345-55. doi: 10.1084/jem.183.4.1345.
10
Selective development of T helper (Th)2 cells induced by continuous administration of low dose soluble proteins to normal and beta(2)-microglobulin-deficient BALB/c mice.通过持续给正常和β2-微球蛋白缺陷的BALB/c小鼠注射低剂量可溶性蛋白诱导T辅助（Th）2细胞的选择性发育。
J Exp Med. 1996 Feb 1;183(2):485-97. doi: 10.1084/jem.183.2.485.
Zotero 插件