介导对蛋白质和合成多肽抗原的迟发型超敏反应的效应T细胞的体外和体内诱导 - Suppr

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超能文献

In vitro and in vivo induction of effector T cells mediating DTH responses to a protein and a synthetic polypeptide antigen.

作者信息

Eshhar Z, Strassmann G, Waks T, Mozes E

出版信息

Cell Immunol. 1979 Oct;47(2):378-89. doi: 10.1016/0008-8749(79)90347-2.

DOI:10.1016/0008-8749(79)90347-2

PMID:90558

Abstract

摘要

相似文献

In vitro and in vivo induction of effector T cells mediating DTH responses to a protein and a synthetic polypeptide antigen.介导对蛋白质和合成多肽抗原的迟发型超敏反应的效应T细胞的体外和体内诱导

Cell Immunol. 1979 Oct;47(2):378-89. doi: 10.1016/0008-8749(79)90347-2.

T cell responses induced by the parenteral injection of antigen-modified syngeneic cells. I. Induction, characterization, and regulation of antigen-specific T helper cells involved in delayed-type hypersensitivity responses.经肠胃外注射抗原修饰的同基因细胞诱导的T细胞应答。I. 参与迟发型超敏反应的抗原特异性辅助性T细胞的诱导、特征及调节。

J Immunol. 1983 Jul;131(1):77-85.

Activation of T cells by glutaraldehyde-fixed erythrocyte antigens: radiosensitivity of cells mediating delayed-type hypersensitivity reactions in the mouse.戊二醛固定红细胞抗原对T细胞的激活：小鼠中介导迟发型超敏反应细胞的放射敏感性

Cell Immunol. 1979 Sep 15;47(1):170-81. doi: 10.1016/0008-8749(79)90325-3.

Genetic regulation of delayed-type hypersensitivity responses to poly(LTyr,LGu)-poly(DLAla)--poly(LLys). I. Expression of the genetic defect at two phases of the immune process.对聚（L-酪氨酸，L-谷氨酸）-聚（D-丙氨酸）--聚（L-赖氨酸）迟发型超敏反应的遗传调控。I. 免疫过程两个阶段遗传缺陷的表达

J Exp Med. 1980 Feb 1;151(2):265-74. doi: 10.1084/jem.151.2.265.

T-T cell interaction in the induction of delayed-type hypersensitivity (DTH) responses: vaccinia virus-reactive helper T cell activity involved in enhanced in vivo induction of DTH responses and its application to augmentation of tumor-specific DTH responses.迟发型超敏反应（DTH）诱导过程中的T - T细胞相互作用：痘苗病毒反应性辅助性T细胞活性参与增强DTH反应的体内诱导及其在增强肿瘤特异性DTH反应中的应用。

J Immunol. 1985 Jan;134(1):108-13.

T cells expressing delayed-type hypersensitivity can be derived from a humorally immune lymphocyte population.表达迟发型超敏反应的T细胞可源自体液免疫淋巴细胞群体。

Eur J Immunol. 1987 Jul;17(7):949-54. doi: 10.1002/eji.1830170709.

Desensitization in mice: T-cell requirement for nonspecific suppression of delayed-type hypersensitivity.小鼠脱敏：迟发型超敏反应非特异性抑制的T细胞需求。

Cell Immunol. 1981 May 15;60(2):308-13. doi: 10.1016/0008-8749(81)90272-0.

Distinct subsets of accessory cells activate Thy-1+ triple negative (CD3-, CD4-, CD8-) cells and Th-1 delayed-type hypersensitivity effector T cells.不同亚群的辅助细胞激活Thy-1+三阴性（CD3-、CD4-、CD8-）细胞和Th-1迟发型超敏反应效应T细胞。

J Immunol. 1991 May 15;146(10):3322-31.

Effects of pertussis toxin (PT) on T-cell populations sensitized for delayed-type hypersensitivity in mice.百日咳毒素（PT）对小鼠中为迟发型超敏反应致敏的T细胞群体的影响。

Cell Immunol. 1984 May;85(2):351-63. doi: 10.1016/0008-8749(84)90249-1.

Induction by killed Listeria monocytogenes of effector T cells mediating delayed-type hypersensitivity but not protection in mice.用灭活的单核细胞增生李斯特菌诱导效应T细胞，该效应T细胞介导小鼠的迟发型超敏反应，但不介导保护作用。

Immunology. 1987 Oct;62(2):241-8.

引用本文的文献

Cellular analysis of specificity of antibodies and of delayed type hypersensitivity responses toward some structurally related synthetic antigens: boosting is determined by specificity of T cells.针对某些结构相关合成抗原的抗体特异性及迟发型超敏反应的细胞分析：增强作用由T细胞特异性决定。

Proc Natl Acad Sci U S A. 1982 Sep;79(18):5636-40. doi: 10.1073/pnas.79.18.5636.

Genetic regulation of delayed-type hypersensitivity responses to poly (Tyr,Glu)-poly(DLAla)--poly(Lys): expression of the genetic defect in the induction and manifestation phases in H-2s and H-2f mice.对聚（酪氨酸，谷氨酸）-聚（D-丙氨酸）-聚（L-赖氨酸）迟发型超敏反应的遗传调控：H-2s和H-2f小鼠诱导期和表现期遗传缺陷的表达

Immunology. 1982 Mar;45(3):405-11.

Elicitation of delayed-type hypersensitivity responses to poly(L-Tyr,LGlu)-poly(DLAla)--poly(LLys) by anti-idiotypic antibodies.抗独特型抗体引发对聚（L-酪氨酸，L-谷氨酸）-聚（D-丙氨酸）-聚（L-赖氨酸）的迟发型超敏反应

J Exp Med. 1980 Nov 1;152(5):1448-52. doi: 10.1084/jem.152.5.1448.

Antiviral response elicited by a completely synthetic antigen with built-in adjuvanticity.由具有内在佐剂活性的完全合成抗原引发的抗病毒反应。

Proc Natl Acad Sci U S A. 1980 Nov;77(11):6769-72. doi: 10.1073/pnas.77.11.6769.

Fine specificity and idiotypic expression of monoclonal antibodies directed against poly(Tyr,Glu)-poly(DLAla)--poly(Lys) and its ordered analogue (Tyr-Tyr-Glu-Glu)-poly(DLAla)--poly(Lys).针对聚（酪氨酸，谷氨酸）-聚（D-丙氨酸）-聚（赖氨酸）及其有序类似物（酪氨酸-酪氨酸-谷氨酸-谷氨酸）-聚（D-丙氨酸）-聚（赖氨酸）的单克隆抗体的精细特异性和独特型表达

Immunology. 1983 May;49(1):9-19.

Mutations at H-2K or H-2D alter immune response phenotype of autoimmune thyroiditis.H-2K或H-2D处的突变会改变自身免疫性甲状腺炎的免疫反应表型。

Immunogenetics. 1982;15(6):625-7. doi: 10.1007/BF00347059.

The inductive requirements for the primary in vitro generation of delayed-type hypersensitivity response to influenza virus in mice.小鼠对流感病毒迟发型超敏反应体外初次产生的诱导要求。

Immunology. 1981 Sep;44(1):17-28.

Specificity of genes controlling immune responsiveness to (T,G)-A--L and (Phe,G)-A--L.控制对（T，G）-A--L和（苯丙氨酸，G）-A--L免疫反应性的基因的特异性

Immunology. 1980 Oct;41(2):339-46.

Efficient genetically controlled formation of antibody to a synthetic antigen [poly(LTyr, LGlu)-poly(DLAla)- -poly(LLys)] covalently bound to a synthetic adjuvant (N-acetylmuramyl-L-alanyl-D-isoglutamine).高效地通过基因控制形成针对与合成佐剂（N-乙酰胞壁酰-L-丙氨酰-D-异谷氨酰胺）共价结合的合成抗原[聚（L-酪氨酸，L-谷氨酸）-聚（D-丙氨酸）-聚（L-赖氨酸）]的抗体。

Proc Natl Acad Sci U S A. 1980 Aug;77(8):4933-7. doi: 10.1073/pnas.77.8.4933.

H-2K mutation controls immune response phenotype of autoimmune thyroiditis. Critical expression of mutant gene product in both thymus and thyroid glands.H-2K突变控制自身免疫性甲状腺炎的免疫反应表型。突变基因产物在胸腺和甲状腺中均有关键表达。

J Exp Med. 1980 Oct 1;152(4):1115-20. doi: 10.1084/jem.152.4.1115.

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