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Tumor necrosis factor (TNF) and nitric oxide (NO) mediate the acute cardiovascular effects of endotoxemia.

作者信息

Odeh M

出版信息

Ann Surg. 1997 Mar;225(3):342. doi: 10.1097/00000658-199703000-00020.

Abstract
摘要

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本文引用的文献

1
The role of tumor necrosis factor and nitric oxide in the acute cardiovascular response to endotoxin.
Ann Surg. 1996 Jan;223(1):63-9. doi: 10.1097/00000658-199601000-00009.
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Cardiac depressant effects of oxygen free radicals.
Angiology. 1993 Apr;44(4):257-70. doi: 10.1177/000331979304400401.
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Tumor necrosis factor-alpha as a myocardial depressant substance.
Int J Cardiol. 1993 Dec 31;42(3):231-8. doi: 10.1016/0167-5273(93)90053-j.
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Enhancement of intracellular glutathione protects endothelial cells against oxidant damage.
Biochem Biophys Res Commun. 1985 Feb 28;127(1):270-6. doi: 10.1016/s0006-291x(85)80154-6.
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Tumor necrosis factor provokes superoxide anion generation from neutrophils.
Biochem Biophys Res Commun. 1986 Jun 30;137(3):1094-100. doi: 10.1016/0006-291x(86)90337-2.
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Effect of oxygen free radicals on cardiovascular function at organ and cellular levels.
Am Heart J. 1989 Jun;117(6):1196-202. doi: 10.1016/0002-8703(89)90396-7.

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