Shriberg L D, Aram D M, Kwiatkowski J
University of Wisconsin-Madison, USA.
J Speech Lang Hear Res. 1997 Apr;40(2):313-37. doi: 10.1044/jslhr.4002.313.
Two prior studies in this series (Shriberg, Aram, & Kwiatkowski, 1997a, 1997b) address the premise that children with developmental apraxia of speech (DAS) can be differentiated from children with speech delay (SD) on the basis of one or more reliable differences in their speech. The first study compared segmental and prosody-voice profiles of a group of 14 children with suspected DAS to profiles of 73 children with SD. Results suggest that the only linguistic domain that differentiates some children with suspected DAS from those with SD is inappropriate stress. The second study cross-validated these findings, using retrospective data from a sample of 20 children with suspected DAS evaluated in a university phonology clinic over a 10-year period. The present study is of particular interest because it cross-validates the prior stress findings, using conversational speech samples from 19 children with suspected DAS provided by five DAS researchers at geographically diverse diagnostic facilities in North America. Summed across the three studies, 52% of 48 eligible samples from 53 children with suspected DAS had inappropriate stress, compared to 10% of 71 eligible samples from 73 age-matched children with speech delay of unknown origin. Discussion first focuses on the implications of stress findings for theories of the origin and nature of DAS. Perspectives in psycholinguistics, neurolinguistics, and developmental biolinguistics lead to five working hypotheses pending validation in ongoing studies: (a) inappropriate stress is a diagnostic marker for at least one subtype of DAS, (b) the psycholinguistic loci of inappropriate stress in this subtype of DAS are in phonological representational processes, (c) the proximal origin of this subtype of DAS is a neurogenically specific deficit, (d) the distal origin of this form of DAS is an inherited genetic polymorphism, and (e) significant differences between acquired apraxia of speech in adults and findings for this subtype of DAS call into question the inference that it is an apractic, motor speech disorder. Concluding discussion considers implications of these findings for research in DAS and for clinical practice.
本系列先前的两项研究(施里伯格、阿拉姆和克维亚特科夫斯基,1997a,1997b)探讨了这样一个前提,即患有发育性言语失用症(DAS)的儿童可以根据其言语中一个或多个可靠差异与言语延迟(SD)儿童区分开来。第一项研究将一组14名疑似DAS儿童的音段和韵律-语音特征与73名SD儿童的特征进行了比较。结果表明,将一些疑似DAS儿童与SD儿童区分开来的唯一语言领域是不适当的重音。第二项研究使用了在10年期间于大学语音诊所评估的20名疑似DAS儿童样本的回顾性数据对这些发现进行了交叉验证。本研究特别引人关注,因为它使用了来自北美不同地理位置诊断机构的五位DAS研究人员提供的19名疑似DAS儿童的对话语音样本,对先前的重音发现进行了交叉验证。在这三项研究中,53名疑似DAS儿童的48个合格样本中有52%存在不适当重音,相比之下,73名年龄匹配、病因不明的言语延迟儿童的71个合格样本中有10%存在不适当重音。讨论首先聚焦于重音发现对DAS起源和本质理论的影响。心理语言学、神经语言学和发育生物语言学的观点引出了五个有待正在进行的研究验证的工作假设:(a)不适当重音是至少一种DAS亚型的诊断标志,(b)这种DAS亚型中不适当重音的心理语言学位点在于音系表征过程,(c)这种DAS亚型的近端起源是神经源性特异性缺陷,(d)这种形式的DAS的远端起源是一种遗传多态性,(e)成人获得性言语失用症与这种DAS亚型的发现之间的显著差异对其是一种失用性运动言语障碍的推断提出了质疑。结论性讨论考虑了这些发现对DAS研究和临床实践的影响。