Greenberg M
Am J Ind Med. 1997 Sep;32(3):309-14. doi: 10.1002/(sici)1097-0274(199709)32:3<309::aid-ajim19>3.0.co;2-z.
Toward the end of the 1920s, technical advances in mining led to an increase in airborne burdens of dust in the South Wales coal mines. This coincided with a dramatic increase in the incidence of disability and death from respiratory disease among the miners. For their condition to be compensable, claimants were required to have worked with rock containing more than 50% 'free silica.' Dr W.R. Jones, a mining geologist, was asked to help obtain compensation for those claimants who could not satisfy the 'free silica' condition. He was unable to identify high-silica rocks where none had been said to exist. He did however, successfully argue the brief against the eminent Professor J.S. Haldane (who was the dominant authority, having had lengthy experience in the field of health and mining), for the fibrous form of sericite being commonly the important agent responsible for pneumoconiosis. As a consequence, the category of miner eligible for compensation was broadened. Evidence was gathered worldwide that supported the hypothesis that silicates and not just crystalline silica could cause pneumoconiosis. Despite the suspicions raised about the special power of mineral fibers during this public debate, some 40 years were to elapse before potential health hazards from fibers other than asbestos were to be taken seriously and investigated.
20世纪20年代末,采矿技术的进步导致南威尔士煤矿空气中的粉尘负担增加。与此同时,矿工中因呼吸系统疾病导致的残疾和死亡发生率急剧上升。为了使他们的病情能够得到赔偿,索赔者被要求曾在含有超过50%“游离二氧化硅”的岩石中工作。采矿地质学家W.R.琼斯博士被要求帮助那些不符合“游离二氧化硅”条件的索赔者获得赔偿。在据称不存在高硅岩石的地方,他无法识别出高硅岩石。然而,他成功地反驳了杰出的J.S.霍尔丹教授(他是该领域的权威,在健康与采矿领域有长期经验),认为绢云母的纤维形态通常是导致尘肺病的重要因素。结果,有资格获得赔偿的矿工类别扩大了。世界各地收集的证据支持了硅酸盐而非仅仅是结晶二氧化硅会导致尘肺病的假设。尽管在这场公开辩论中有人对矿物纤维的特殊危害提出了怀疑,但在石棉以外的纤维潜在健康危害得到认真对待和调查之前,大约又过去了40年。