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因泌尿道感染引发的母体体温过低导致胎儿长期心动过缓。

Prolonged fetal bradycardia secondary to maternal hypothermia in response to urosepsis.

作者信息

Hankins G D, Leicht T, Van Hook J W

机构信息

Department of Obstetrics and Gynecology, University of Texas Medical Branch at Galveston 77555-0587, USA.

出版信息

Am J Perinatol. 1997 Apr;14(4):217-9. doi: 10.1055/s-2007-994130.

Abstract

Fetal bradycardia is a well-known response to maternal hypothermia, as induced at open-heart surgery, but heretofore has not been reported in conjunction with hypothermia from urosepsis. A 24-year-old Vietnamese woman admitted at 33 weeks estimated gestational age with pyelonephritis secondary to Escherichia coli developed several episodes of maternal hypothermia to 35-36 degrees C. During each episode of maternal hypothermia, the baseline fetal heart rate fell to 90-100 bpm, but with retained reactivity. During each episode, maternal vital signs were otherwise stable and oxygen saturation was normal as measured by pulse oximetry. Interpretation of fetal bradycardia during episodes of maternal urosepsis is complex. If seen in conjunction with maternal hypothermia, and in the presence of normal maternal cardiac and respiratory function, bradycardia is unlikely to represent fetal distress.

摘要

胎儿心动过缓是一种已知的对母体低温的反应,如在心脏直视手术中诱发的那样,但迄今为止尚未有与泌尿道感染所致低温相关的报道。一名24岁的越南女性,孕33周估计孕周时因大肠杆菌引起的肾盂肾炎入院,出现了几次母体体温降至35 - 36摄氏度的情况。在每次母体体温过低发作期间,胎儿心率基线降至90 - 100次/分钟,但仍保持反应性。在每次发作期间,母体生命体征其他方面稳定,经脉搏血氧饱和度测定法测得的氧饱和度正常。在母体泌尿道感染发作期间对胎儿心动过缓的解读很复杂。如果与母体低温同时出现,且母体心脏和呼吸功能正常,心动过缓不太可能代表胎儿窘迫。

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