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人工诱导贫血对热应激时汗腺分泌及皮肤血流反应的影响。

Effects of artificially-induced anaemia on sudomotor and cutaneous blood flow responses to heat stress.

作者信息

Taylor N A, Patterson M J, Cotter J D, Macfarlane D J

机构信息

Department of Biomedical Science, University of Wollongong, NSW, Australia.

出版信息

Eur J Appl Physiol Occup Physiol. 1997;76(4):380-6. doi: 10.1007/s004210050265.

Abstract

The influence of artificially induced anaemia on thermal strain was evaluated in trained males. Heat stress trials (38.6 degrees C, water vapour pressure 2.74 kPa) performed at the same absolute work rates [20 min of seated rest, 20 min of cycling at 30% peak aerobic power (VO2pcak), and 20 min cycling at 45% VO2peak] were completed before (HST1) and 3-5 days after 3 units of whole blood were withdrawn (HST2). Mild anaemia did not elevate thermal strain between trials, with auditory canal temperatures terminating at 38.5 degrees C [(0.16), HST1] and 38.6 degrees C [(0.13), HST2; P > 0.05]. Given that blood withdrawal reduced aerobic power by 16%, this observation deviates from the close association often observed between core temperature and relative exercise intensity. During HST2, the absolute and integrated forearm sweat rate (mSW) exceeded control levels during exercise (P < 0.05), while a suppression of forehead mSW occurred (P < 0.05). These observations are consistent with a possible peripheral redistribution of sweat secretion. It was concluded that this level of artificially induced anaemia did not impact upon heat strain during a 60-min heat stress test.

摘要

在受过训练的男性中评估了人工诱导贫血对热应激的影响。在抽取3单位全血前(热应激试验1,HST1)和抽取后3 - 5天(热应激试验2,HST2),以相同的绝对工作速率[20分钟坐姿休息、以30%峰值有氧功率(VO₂峰值)骑行20分钟、以45%VO₂峰值骑行20分钟]进行热应激试验(38.6℃,水汽压2.74kPa)。轻度贫血在两次试验之间未增加热应激,外耳道温度在HST1时终止于38.5℃[(0.16)],在HST2时终止于38.6℃[(0.13)];P>0.05。鉴于采血使有氧功率降低了16%,这一观察结果偏离了通常在核心温度与相对运动强度之间观察到的紧密关联。在HST2期间,运动期间前臂绝对和累计出汗率(mSW)超过对照水平(P<0.05),而前额mSW出现抑制(P<0.05)。这些观察结果与出汗分泌可能的外周重新分布一致。得出的结论是,在60分钟的热应激试验期间,这种程度的人工诱导贫血不会影响热应激。

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