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Tumor necrosis factor in septic shock and multiple system trauma.

作者信息

Dellinger R P

出版信息

Crit Care Med. 1997 Nov;25(11):1771-3. doi: 10.1097/00003246-199711000-00005.

DOI:10.1097/00003246-199711000-00005
PMID:9366750
Abstract
摘要

相似文献

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Tumor necrosis factor in septic shock and multiple system trauma.脓毒性休克和多系统创伤中的肿瘤坏死因子
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Patterns of cytokine evolution (tumor necrosis factor-alpha and interleukin-6) after septic shock, hemorrhagic shock, and severe trauma.脓毒性休克、失血性休克和严重创伤后细胞因子(肿瘤坏死因子-α和白细胞介素-6)的演变模式
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Immunological status of septic and trauma patients. I. High tumor necrosis factor alpha serum levels in septic and trauma patients are not responsible for increased mortality; a prognostic value of serum interleukin 6.脓毒症和创伤患者的免疫状态。I. 脓毒症和创伤患者血清肿瘤坏死因子α水平升高并非死亡率增加的原因;血清白细胞介素6的预后价值
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Proinflammatory and anti-inflammatory cytokines as mediators in the pathogenesis of septic shock.促炎和抗炎细胞因子作为脓毒性休克发病机制中的介质。
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Transforming growth factor-beta1 blocks in vitro cardiac myocyte depression induced by tumor necrosis factor-alpha, interleukin-1beta, and human septic shock serum.转化生长因子-β1可阻断肿瘤坏死因子-α、白细胞介素-1β和人类感染性休克血清诱导的体外心肌细胞抑制。
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Severe human septic shock involves more than tumor necrosis factor.严重的人类脓毒性休克涉及的不止是肿瘤坏死因子。
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Modulation of cytokine responses in sepsis.
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The mRNA expression patterns of tumor necrosis factor-alpha and TNFR-I in some vital organs after thermal injury.热损伤后某些重要器官中肿瘤坏死因子-α和肿瘤坏死因子受体-I的mRNA表达模式
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Increased plasma levels of corticosterone and prolactin and decreased T3 and T4 levels in short-term prehepatic portal hypertension in rats.
大鼠短期肝前性门静脉高压时血浆皮质酮和催乳素水平升高,T3和T4水平降低。
Dig Dis Sci. 2000 Sep;45(9):1865-71. doi: 10.1023/a:1005588918516.
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Early activation of hepatic NFkappaB and NF-IL6 in polymicrobial sepsis correlates with bacteremia, cytokine expression, and mortality.多微生物脓毒症中肝脏NFκB和NF-IL6的早期激活与菌血症、细胞因子表达及死亡率相关。
Ann Surg. 1999 Jul;230(1):95-104. doi: 10.1097/00000658-199907000-00014.