Wilson A J, Furness J B, Costa M
Neurosci Lett. 1979 Oct;14(2-3):303-8. doi: 10.1016/0304-3940(79)96165-2.
A positive uranaffin reaction was observed in the small (40-60 nm) diameter vesicles of some intestinal axons. There was no change in the number of reactive axons or the intensity of reaction after reserpine (5 mg/kg) or after interruption of axons reaching the intestine through the mesentery. The axons were found in the myenteric, submucous and deep muscular plexuses and in the circular muscle. Some uranaffin-positive axons formed synapses with neurons of the myenteric and submucous plexuses. It is concluded that these axons are not noradrenergic. The axons must represent one of the several nerve types which are known to be intrinsic to the intestine, but are as yet unidentified at an ultrastructural level. If, as has been postulated, the reaction localizes amine storage vesicles, the uranaffin-positive axons are probably the intrinsic amine-handling axons previously demonstrated histochemically.
在一些肠轴突直径较小（40 - 60纳米）的囊泡中观察到了阳性铀萘酚反应。给予利血平（5毫克/千克）后，或通过肠系膜切断到达肠道的轴突后，反应性轴突的数量或反应强度均未发生变化。这些轴突见于肌间神经丛、黏膜下神经丛和深部肌丛以及环行肌中。一些铀萘酚阳性轴突与肌间神经丛和黏膜下神经丛的神经元形成突触。得出的结论是，这些轴突不是去甲肾上腺素能的。这些轴突必定代表了已知为肠道固有但在超微结构水平上尚未明确的几种神经类型之一。如果如所推测的那样，该反应定位胺储存囊泡，那么铀萘酚阳性轴突可能是先前经组织化学证明的固有胺处理轴突。
