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Profiles of brain glycosidase activity in cuprizone-fed Syrian hamsters and in scrapie-affected mice, rats, Chinese hamsters and Syrian hamsters.

作者信息

Kimberlin R H, Collis S C, Walker C A

出版信息

J Comp Pathol. 1976 Jan;86(1):135-42. doi: 10.1016/0021-9975(76)90038-4.

DOI:10.1016/0021-9975(76)90038-4
PMID:943426
Abstract
摘要

相似文献

1
Profiles of brain glycosidase activity in cuprizone-fed Syrian hamsters and in scrapie-affected mice, rats, Chinese hamsters and Syrian hamsters.
J Comp Pathol. 1976 Jan;86(1):135-42. doi: 10.1016/0021-9975(76)90038-4.
2
The effects of cuprizone toxicity on the incubation period of scrapie in mice.
J Comp Pathol. 1976 Jul;86(3):489-96. doi: 10.1016/0021-9975(76)90017-7.
3
A comparison of the biochemical changes induced in mouse brain cuprizone toxicity and by scrapie infection.
J Comp Pathol. 1974 Apr;84(2):263-70. doi: 10.1016/0021-9975(74)90067-x.
4
Retinal degeneration during clinical scrapie encephalopathy in hamsters.仓鼠临床瘙痒病脑病期间的视网膜变性。
J Comp Neurol. 1982 Feb 20;205(2):153-60. doi: 10.1002/cne.902050206.
5
Glycosidase histochemistry in normal and scrapie mice, rats, sheep and goats.正常及患瘙痒病的小鼠、大鼠、绵羊和山羊的糖苷酶组织化学。
J Comp Pathol. 1968 Jan;78(1):43-52. doi: 10.1016/0021-9975(68)90111-4.
6
Clinical and histological recovery from the scrapie-like spongiform encephalopathy produced in mice by feeding them with cuprizone.通过给小鼠喂食铜螯合剂所引发的类羊瘙痒症海绵状脑病的临床及组织学恢复情况。
J Pathol. 1973 Mar;109(3):245-50. doi: 10.1002/path.1711090310.
7
Permeability of blood vessels in mice affected with scrapie or fed with cuprizone.
J Comp Pathol. 1973 Oct;83(4):461-6. doi: 10.1016/0021-9975(73)90003-0.
8
Brain glycosidases in Creutzfeldt-Jakob disease.克雅氏病中的脑糖苷酶
J Neurol Sci. 1981 Feb;49(2):325-8. doi: 10.1016/0022-510x(81)90088-5.
9
Cuprizone neurotoxicity in the rat: morphologic observations.
J Neurol Sci. 1988 Apr;84(2-3):223-37. doi: 10.1016/0022-510x(88)90127-x.
10
Enzymatic changes in scrapie agent-infected mouse brain.瘙痒病病原体感染的小鼠大脑中的酶变化
Am J Vet Res. 1971 Jun;32(6):959-66.

引用本文的文献

1
Oligodendrocyte death and myelin loss in the cuprizone model: an updated overview of the intrinsic and extrinsic causes of cuprizone demyelination.寡突胶质细胞死亡和髓鞘丢失在铜诱导模型中的作用:铜诱导脱髓鞘的内在和外在原因的最新综述。
Mol Neurodegener. 2022 May 7;17(1):34. doi: 10.1186/s13024-022-00538-8.
2
Heterogeneity in oligodendroglia: Is it relevant to mouse models and human disease?少突胶质细胞的异质性:它与小鼠模型和人类疾病相关吗?
J Neurosci Res. 2016 Dec;94(12):1421-1433. doi: 10.1002/jnr.23900. Epub 2016 Aug 25.
3
Glial response during cuprizone-induced de- and remyelination in the CNS: lessons learned.
中枢神经系统中海洛因诱导的脱髓鞘和髓鞘再生过程中的神经胶质反应:经验教训。
Front Cell Neurosci. 2014 Mar 13;8:73. doi: 10.3389/fncel.2014.00073. eCollection 2014.