Macrophage receptors for Mycobacterium tuberculosis.
作者信息
Ernst J D
机构信息
Department of Medicine, University of California, San Francisco, 94143-0868, USA.
出版信息
Infect Immun. 1998 Apr;66(4):1277-81. doi: 10.1128/IAI.66.4.1277-1281.1998.
Abstract
摘要
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Macrophage receptors for Mycobacterium tuberculosis.结核分枝杆菌的巨噬细胞受体
Infect Immun. 1998 Apr;66(4):1277-81. doi: 10.1128/IAI.66.4.1277-1281.1998.
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Selective receptor blockade during phagocytosis does not alter the survival and growth of Mycobacterium tuberculosis in human macrophages.吞噬作用期间的选择性受体阻断不会改变结核分枝杆菌在人类巨噬细胞中的存活和生长。
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Mycobacterium tuberculosis uptake by recipient host macrophages is influenced by environmental conditions in the granuloma of the infectious individual and is associated with impaired production of interleukin-12 and tumor necrosis factor alpha.受感染个体肉芽肿中的环境条件会影响宿主巨噬细胞对结核分枝杆菌的摄取，且这与白细胞介素-12和肿瘤坏死因子α的产生受损有关。
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本文引用的文献
1
Complement receptor type 1 (CR1, CD35) is a receptor for C1q.补体受体1（CR1，CD35）是C1q的受体。
Immunity. 1997 Sep;7(3):345-55. doi: 10.1016/s1074-7613(00)80356-8.
2
Nonopsonic binding of Mycobacterium tuberculosis to complement receptor type 3 is mediated by capsular polysaccharides and is strain dependent.结核分枝杆菌与3型补体受体的非调理素结合由荚膜多糖介导，且具有菌株依赖性。
Infect Immun. 1997 Oct;65(10):4258-66. doi: 10.1128/iai.65.10.4258-4266.1997.
3
Interaction of Mycobacterium avium with environmental amoebae enhances virulence.鸟分枝杆菌与环境中的变形虫相互作用会增强其毒力。
Infect Immun. 1997 Sep;65(9):3759-67. doi: 10.1128/iai.65.9.3759-3767.1997.
4
Surfactant protein A (SP-A) mediates attachment of Mycobacterium tuberculosis to murine alveolar macrophages.表面活性蛋白A（SP-A）介导结核分枝杆菌与小鼠肺泡巨噬细胞的附着。
Am J Respir Cell Mol Biol. 1997 Aug;17(2):209-17. doi: 10.1165/ajrcmb.17.2.2469.
5
A macrophage invasion mechanism of pathogenic mycobacteria.致病性分枝杆菌的巨噬细胞侵袭机制。
Science. 1997 Aug 22;277(5329):1091-3. doi: 10.1126/science.277.5329.1091.
6
Surrogate marker of preclinical tuberculosis in human immunodeficiency virus infection: antibodies to an 88-kDa secreted antigen of Mycobacterium tuberculosis.人类免疫缺陷病毒感染中临床前结核病的替代标志物：针对结核分枝杆菌88 kDa分泌抗原的抗体
J Infect Dis. 1997 Jul;176(1):133-43. doi: 10.1086/514015.
7
SP-A enhances uptake of bacillus Calmette-Guérin by macrophages through a specific SP-A receptor.表面活性蛋白A通过特定的表面活性蛋白A受体增强巨噬细胞对卡介苗的摄取。
Am J Physiol. 1997 May;272(5 Pt 1):L989-95. doi: 10.1152/ajplung.1997.272.5.L989.
8
Arrest of mycobacterial phagosome maturation is caused by a block in vesicle fusion between stages controlled by rab5 and rab7.分枝杆菌吞噬体成熟的停滞是由rab5和rab7控制阶段之间的囊泡融合受阻所致。
J Biol Chem. 1997 May 16;272(20):13326-31. doi: 10.1074/jbc.272.20.13326.
9
Growth within macrophages increases the efficiency of Mycobacterium avium in invading other macrophages by a complement receptor-independent pathway.巨噬细胞内的生长通过不依赖补体受体的途径提高了鸟分枝杆菌侵入其他巨噬细胞的效率。
Infect Immun. 1997 May;65(5):1916-25. doi: 10.1128/iai.65.5.1916-1925.1997.
10
The mannose receptor delivers lipoglycan antigens to endosomes for presentation to T cells by CD1b molecules.甘露糖受体将脂多糖抗原递送至内体，以便通过CD1b分子呈递给T细胞。
Immunity. 1997 Feb;6(2):187-97. doi: 10.1016/s1074-7613(00)80425-2.
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