一种源自类风湿性关节炎患者的新型滑膜细胞系（RAMAK-1）。 - Suppr

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超能文献

A novel synoviocyte line (RAMAK-1), derived from a patient with rheumatoid arthritis.

作者信息

Kawakami M, Matsukuma S, Hara M, Hidaka T, Suzuki K, Kitani A, Hiroi S, Ishizuka T, Matsuki Y, Nakamura H

出版信息

In Vitro Cell Dev Biol Anim. 1998 Feb;34(2):85-7. doi: 10.1007/s11626-998-0086-x.

DOI:10.1007/s11626-998-0086-x

PMID:9542641

Abstract

摘要

相似文献

A novel synoviocyte line (RAMAK-1), derived from a patient with rheumatoid arthritis.一种源自类风湿性关节炎患者的新型滑膜细胞系（RAMAK-1）。

In Vitro Cell Dev Biol Anim. 1998 Feb;34(2):85-7. doi: 10.1007/s11626-998-0086-x.

Grafting of fibroblasts isolated from the synovial membrane of rheumatoid arthritis (RA) patients induces chronic arthritis in SCID mice-A novel model for studying the arthritogenic role of RA fibroblasts in vivo.从类风湿性关节炎（RA）患者滑膜中分离出的成纤维细胞移植可在SCID小鼠中诱发慢性关节炎——一种用于在体内研究RA成纤维细胞致关节炎作用的新模型。

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Stimulation of collagenase 3 expression in synovial fibroblasts of patients with rheumatoid arthritis by contact with a three-dimensional collagen matrix or with normal cartilage when coimplanted in NOD/SCID mice.类风湿性关节炎患者的滑膜成纤维细胞在与三维胶原基质接触或与正常软骨共同植入NOD/SCID小鼠时，胶原酶3表达受到刺激。

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FLIP and MAPK play crucial roles in the MLN51-mediated hyperproliferation of fibroblast-like synoviocytes in the pathogenesis of rheumatoid arthritis.在类风湿性关节炎发病机制中，FLIP和丝裂原活化蛋白激酶（MAPK）在MLN51介导的成纤维样滑膜细胞过度增殖中发挥关键作用。

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Acute serum amyloid A induces migration, angiogenesis, and inflammation in synovial cells in vitro and in a human rheumatoid arthritis/SCID mouse chimera model.急性血清淀粉样蛋白 A 在体外诱导滑膜细胞迁移、血管生成和炎症，并在人类类风湿关节炎/SCID 嵌合小鼠模型中诱导。

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引用本文的文献

ECV304 (endothelial) is really T24 (bladder carcinoma): cell line cross- contamination at source.ECV304（内皮细胞系）实际上是T24（膀胱癌细胞系）：细胞系在来源处就存在交叉污染。

In Vitro Cell Dev Biol Anim. 1999 Nov-Dec;35(10):558-9. doi: 10.1007/s11626-999-0091-8.

Hyaluronan production in human rheumatoid fibroblastic synovial lining cells is increased by interleukin 1 beta but inhibited by transforming growth factor beta 1.白细胞介素1β可增加人类风湿性成纤维细胞滑膜衬里细胞中的透明质酸生成，但转化生长因子β1则对其有抑制作用。

Ann Rheum Dis. 1998 Oct;57(10):602-5. doi: 10.1136/ard.57.10.602.

本文引用的文献

Synthesis of hyaluronic acid by human synovial tissue slices.人滑膜组织切片对透明质酸的合成

Science. 1957 Jun 28;125(3261):1300. doi: 10.1126/science.125.3261.1300.

Synovial fibroblast-like cell transfection with the SV40 large T antigen induces a transformed phenotype and permits transient tumor formation in immunodeficient mice.用SV40大T抗原转染滑膜成纤维样细胞可诱导其转化表型，并使其在免疫缺陷小鼠中形成短暂性肿瘤。

J Rheumatol. 1994 Aug;21(8):1409-19.

Intermediate filaments in synovial lining cells in rheumatoid arthritis and other arthritides are of vimentin type.类风湿关节炎及其他关节炎中滑膜衬里细胞的中间丝属于波形蛋白类型。

Ann Rheum Dis. 1982 Feb;41(1):74-7. doi: 10.1136/ard.41.1.74.

A monoclonal antibody reactive with human peripheral blood monocytes.一种与人外周血单核细胞反应的单克隆抗体。

J Immunol. 1980 Apr;124(4):1943-8.

Human mononuclear phagocyte differentiation antigens. I. Patterns of antigenic expression on the surface of human monocytes and macrophages defined by monoclonal antibodies.人类单核吞噬细胞分化抗原。I. 单克隆抗体所定义的人类单核细胞和巨噬细胞表面抗原表达模式。

J Immunol. 1983 Jan;130(1):145-52.

Ultrastructure of synovial membrane in rheumatoid arthritis.类风湿关节炎滑膜的超微结构

Ann Rheum Dis. 1967 Sep;26(5):426-43. doi: 10.1136/ard.26.5.426.

Synovial giant cells in rheumatoid arthritis.类风湿关节炎中的滑膜巨细胞。

Am J Pathol. 1966 Nov;49(5):931-54.

Identification of tissue histiocytes on paraffin sections by a new monoclonal antibody.用一种新型单克隆抗体在石蜡切片上鉴定组织组织细胞

J Histochem Cytochem. 1987 Nov;35(11):1217-26. doi: 10.1177/35.11.3309045.

Chronic inflammatory arthropathy associated with HTLV-I.

Lancet. 1989 Feb 25;1(8635):441. doi: 10.1016/s0140-6736(89)90038-x.

Expression of intermediate filaments (IF) in tissues and cultured cells.

Int J Dev Biol. 1989 Mar;33(1):55-61.

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