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Fas-induced apoptosis.

作者信息

Nagata S

机构信息

The Department of Genetics, Osaka University Medical School, and Osaka Bioscience Institute, Yamadaoka, Suita.

出版信息

Intern Med. 1998 Feb;37(2):179-81. doi: 10.2169/internalmedicine.37.179.

DOI:10.2169/internalmedicine.37.179
PMID:9550601
Abstract
摘要

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Fas-induced apoptosis.Fas诱导的细胞凋亡。
Intern Med. 1998 Feb;37(2):179-81. doi: 10.2169/internalmedicine.37.179.
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Cell death induced by the Fas/Fas ligand pathway and its role in pathology.Fas/Fas配体途径诱导的细胞死亡及其在病理学中的作用。
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B-Myb overexpression results in activation and increased Fas/Fas ligand-mediated cytotoxicity of T and NK cells.B-Myb过表达导致T细胞和NK细胞的激活以及Fas/Fas配体介导的细胞毒性增加。
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Induction of Jurkat T-cell apoptosis by Fas ligand-transfected endothelial cells.Fas配体转染的内皮细胞诱导Jurkat T细胞凋亡
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Zeta-associated protein of 70 kDa (ZAP-70), but not Syk, tyrosine kinase can mediate apoptosis of T cells through the Fas/Fas ligand, caspase-8 and caspase-3 pathways.70千道尔顿的ζ相关蛋白（ZAP-70）而非脾酪氨酸激酶（Syk），可通过Fas/Fas配体、半胱天冬酶-8和半胱天冬酶-3途径介导T细胞凋亡。
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Soluble Fas modulates interferon responsiveness.可溶性Fas调节干扰素反应性。
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Fas and the immunologically underprivileged intestine.Fas与免疫特惠的肠道
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Fibronectin-associated Fas ligand rapidly induces opposing and time-dependent effects on the activation and apoptosis of T cells.纤连蛋白相关的Fas配体可迅速对T细胞的激活和凋亡产生相反且依赖时间的效应。
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Fas ligand--caught between Scylla and Charybdis.Fas配体——腹背受敌。
Nat Med. 1998 Jan;4(1):21-2. doi: 10.1038/nm0198-021.

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